Acidosis, Renal Tubular

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  Subject Areas on Research

  • Alkali action on the urinary crystallization of calcium salts: contrasting responses to sodium citrate and potassium citrate. 
  • Arthrogryposis, renal tubular acidosis and cholestasis syndrome: spectrum of the clinical manifestations. 
  • Evaluating a critically ill patient with metabolic acidosis: the ifosfamide paradigm. 
  • Hypercalciuria and altered intestinal calcium absorption occurring independently of vitamin D in incomplete distal renal tubular acidosis. 
  • Localization and regulation of the ATP6V0A4 (a4) vacuolar H+-ATPase subunit defective in an inherited form of distal renal tubular acidosis. 
  • Localization of a gene for autosomal recessive distal renal tubular acidosis with normal hearing (rdRTA2) to 7q33-34. 
  • Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness. 
  • Pediatric urolithiasis. 
  • Prevention of recurrent calcium stone formation with potassium citrate therapy in patients with distal renal tubular acidosis. 
  • Renal Tubular Acidosis. 
  • Role of stone analysis in metabolic evaluation and medical treatment of nephrolithiasis. 
  • Safe use of raltegravir and sirolimus in an HIV-infected patient with renal impairment after orthotopic liver transplantation. 
  • Symposium on renal lithiasis. The challenge of calculi in children. 
  • The B1-subunit of the H(+) ATPase is required for maximal urinary acidification. 
  • The renal excretion of hydrogen ion in renal tubular acidosis. I. quantitative assessment of the response to ammonium chloride as an acid load. 
  • The renal excretion of hydrogen ion in renal tubular acidosis. II. Quantitative response to the carbonic anhydrase inhibitor, acetazolamide. 
  • Toluene-induced renal tubular acidosis: an easily missed cause of hypokalemic paralysis. 
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  Keywords of People

  • Preminger, Glenn Michael, James F. Glenn, M.D. Distinguished Professor of Urology, Surgery