Subject Areas on Research
- 2',3'-dideoxy-beta-L-5-fluorocytidine inhibits duck hepatitis B virus reverse transcription and suppresses viral DNA synthesis in hepatocytes, both in vitro and in vivo.
- A high-quality genome and comparison of short- versus long-read transcriptome of the palaearctic duck Aythya fuligula (tufted duck).
- A new duck genome reveals conserved and convergently evolved chromosome architectures of birds and mammals.
- Accumulation of environmental contaminants in wood duck (Aix sponsa) eggs, with emphasis on polychlorinated dibenzo-p-dioxins and polychlorinated dibenzofurans.
- Adrenergic receptors: molecular mechanisms of clinically relevant regulation.
- Convergent effects on cell signaling mechanisms mediate the actions of different neurobehavioral teratogens: alterations in cholinergic regulation of protein kinase C in chick and avian models.
- Design, synthesis and structure relationships of new N,N',N",N"'-tetrakis (omega-amino alkyl) tetraazamacrocycles.
- Effects of DDT on eggshell quality and calcium adenosine triphosphatase.
- Effects of cadmium ingestion and food restriction on energy metabolism and tissue metal concentrations in mallard ducks (Anas platyrhynchos).
- Effects of liver growth factors on hepadnavirus replication in chronically infected duck hepatocytes.
- Embryo toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin to the wood duck (Aix sponsa).
- Enhanced duck hepatitis B virus gene expression following aflatoxin B1 exposure.
- In vitro inhibition of microsomal calcium atpase from eggshell gland of mallard duck.
- Inhibitory effect of 2'-fluoro-5-methyl-beta-L-arabinofuranosyl-uracil on duck hepatitis B virus replication.
- Initial amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of embryonic duck hepatocytes is increased by cell cycle progression.
- Isolation of Campylobacter fetus subsp. jejuni from migratory waterfowl.
- Phorbol diesters promote beta-adrenergic receptor phosphorylation and adenylate cyclase desensitization in duck erythrocytes.
- Residues critical for duck hepatitis B virus neutralization are involved in host cell interaction.
- Respiration during flight in birds.
- Salt stress increases abundance and glycosylation of CFTR localized at apical surfaces of salt gland secretory cells.
- Selenium toxicity: cause and effects in aquatic birds.
- Sublethal effects of cadmium ingestion on mallard ducks.
- The SATE pronucleotide approach applied to acyclovir: part II. Effects of bis(SATE)phosphotriester derivatives of acyclovir on duck hepatitis B virus replication in vitro and in vivo.
- n-Butyrate, a cell cycle blocker, inhibits early amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of duck hepatocytes.