Ducks

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  Subject Areas on Research

  • 2',3'-dideoxy-beta-L-5-fluorocytidine inhibits duck hepatitis B virus reverse transcription and suppresses viral DNA synthesis in hepatocytes, both in vitro and in vivo. 
  • A high-quality genome and comparison of short- versus long-read transcriptome of the palaearctic duck Aythya fuligula (tufted duck). 
  • A new duck genome reveals conserved and convergently evolved chromosome architectures of birds and mammals. 
  • Accumulation of environmental contaminants in wood duck (Aix sponsa) eggs, with emphasis on polychlorinated dibenzo-p-dioxins and polychlorinated dibenzofurans. 
  • Adrenergic receptors: molecular mechanisms of clinically relevant regulation. 
  • Convergent effects on cell signaling mechanisms mediate the actions of different neurobehavioral teratogens: alterations in cholinergic regulation of protein kinase C in chick and avian models. 
  • Design, synthesis and structure relationships of new N,N',N",N"'-tetrakis (omega-amino alkyl) tetraazamacrocycles. 
  • Effects of DDT on eggshell quality and calcium adenosine triphosphatase. 
  • Effects of cadmium ingestion and food restriction on energy metabolism and tissue metal concentrations in mallard ducks (Anas platyrhynchos). 
  • Effects of liver growth factors on hepadnavirus replication in chronically infected duck hepatocytes. 
  • Embryo toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin to the wood duck (Aix sponsa). 
  • Enhanced duck hepatitis B virus gene expression following aflatoxin B1 exposure. 
  • In vitro inhibition of microsomal calcium atpase from eggshell gland of mallard duck. 
  • Inhibitory effect of 2'-fluoro-5-methyl-beta-L-arabinofuranosyl-uracil on duck hepatitis B virus replication. 
  • Initial amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of embryonic duck hepatocytes is increased by cell cycle progression. 
  • Isolation of Campylobacter fetus subsp. jejuni from migratory waterfowl. 
  • Phorbol diesters promote beta-adrenergic receptor phosphorylation and adenylate cyclase desensitization in duck erythrocytes. 
  • Residues critical for duck hepatitis B virus neutralization are involved in host cell interaction. 
  • Respiration during flight in birds. 
  • Salt stress increases abundance and glycosylation of CFTR localized at apical surfaces of salt gland secretory cells. 
  • Selenium toxicity: cause and effects in aquatic birds. 
  • Sublethal effects of cadmium ingestion on mallard ducks. 
  • The SATE pronucleotide approach applied to acyclovir: part II. Effects of bis(SATE)phosphotriester derivatives of acyclovir on duck hepatitis B virus replication in vitro and in vivo. 
  • n-Butyrate, a cell cycle blocker, inhibits early amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of duck hepatocytes.