Epilepsy, Absence
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Subject Areas on Research
- A nineteen-year-old man with altered mental status.
- Age-related relationship between mRNA expression of GABA(B) receptors and calcium channel beta4 subunits in cacnb4lh mice.
- Animal models of nonconvulsive status epilepticus.
- Characterization of the antiabsence effects of SCH 50911, a GABA-B receptor antagonist, in the lethargic mouse, gamma-hydroxybutyrate, and pentylenetetrazole models.
- Differential effects mediated by GABAA receptors in thalamic nuclei in lh/lh model of absence seizures.
- Efficacy of the ketogenic diet in Lennox-Gastaut syndrome: a retrospective review of one institution's experience and summary of the literature.
- Emergency management of the patient with seizures. 1. Classification and treatment system, common seizure disorders.
- Excitatory but not inhibitory synaptic transmission is reduced in lethargic (Cacnb4(lh)) and tottering (Cacna1atg) mouse thalami.
- Focal cerebral metabolic abnormality in a patient with continuous spike waves during slow-wave sleep.
- GABAB receptor-mediated effects in synaptosomes of lethargic (lh/lh) mice.
- GABAB-activated gK+ in thalamic neurons in the lethargic (lh/lh) mouse model of generalized absence seizures.
- Generalized epilepsies: emerging insights into cellular and genetic mechanisms.
- Glutamate decarboxylase isoforms in thalamic nuclei in lethargic mouse model of absence seizures.
- Increased number of GABAB receptors in the lethargic (lh/lh) mouse model of absence epilepsy.
- Lamotrigine in absence and primary generalized epilepsies.
- Models of primary generalized epilepsy.
- Neural network of structures in which GABAB receptors regulate absence seizures in the lethargic (lh/lh) mouse model.
- Non-convulsive status epilepticus: usefulness of clinical features in selecting patients for urgent EEG.
- Oculogyric crises secondary to lamotrigine overdosage.
- Positron emission tomography in pediatric epilepsy.
- Psychogenic seizures: video telemetry observations in 27 patients.
- Stargazing nets new calcium channel subunit.
- Studies of the lethargic (lh/lh) mouse model of absence seizures: regulatory mechanisms and identification of the lh gene.
- The role of GABAB mechanisms in animal models of absence seizures.
- The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice.
- The utility of the lh/lh mutant mouse as a animal model of human absence epilepsy.
- Utility of the lethargic (lh/lh) mouse model of absence seizures in predicting the effects of lamotrigine, vigabatrin, tiagabine, gabapentin, and topiramate against human absence seizures.