Infectious Anemia Virus, Equine

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  Subject Areas on Research

  • A functional genetic approach suggests a novel interaction between the human immunodeficiency virus type 1 (HIV-1) Tat protein and HIV-1 TAR RNA in vivo. 
  • An in vitro transcription system that recapitulates equine infectious anemia virus tat-mediated inhibition of human immunodeficiency virus type 1 Tat activity demonstrates a role for positive transcription elongation factor b and associated proteins in the mechanism of Tat activation. 
  • Analysis of neuronal proliferation, migration and differentiation in the postnatal brain using equine infectious anemia virus-based lentiviral vectors. 
  • Canine cyclin T1 rescues equine infectious anemia virus tat trans-activation in human cells. 
  • Equine infectious anemia virus resists the antiretroviral activity of equine APOBEC3 proteins through a packaging-independent mechanism. 
  • Genetic analysis of the cofactor requirement for human immunodeficiency virus type 1 Tat function. 
  • Highly divergent lentiviral Tat proteins activate viral gene expression by a common mechanism. 
  • Identification of the activation domain of equine infectious anemia virus rev. 
  • Platelets from thrombocytopenic ponies acutely infected with equine infectious anemia virus are activated in vivo and hypofunctional. 
  • Progress in vaccines against AIDS. 
  • Structural features of the trans-activation response RNA element of equine infectious anemia virus. 
  • The structural polypeptides of equine infections anemia virus. 
  • The surface envelope protein gene region of equine infectious anemia virus is not an important determinant of tropism in vitro.