Infectious Anemia Virus, Equine
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Subject Areas on Research
- A functional genetic approach suggests a novel interaction between the human immunodeficiency virus type 1 (HIV-1) Tat protein and HIV-1 TAR RNA in vivo.
- An in vitro transcription system that recapitulates equine infectious anemia virus tat-mediated inhibition of human immunodeficiency virus type 1 Tat activity demonstrates a role for positive transcription elongation factor b and associated proteins in the mechanism of Tat activation.
- Analysis of neuronal proliferation, migration and differentiation in the postnatal brain using equine infectious anemia virus-based lentiviral vectors.
- Canine cyclin T1 rescues equine infectious anemia virus tat trans-activation in human cells.
- Equine infectious anemia virus resists the antiretroviral activity of equine APOBEC3 proteins through a packaging-independent mechanism.
- Genetic analysis of the cofactor requirement for human immunodeficiency virus type 1 Tat function.
- Highly divergent lentiviral Tat proteins activate viral gene expression by a common mechanism.
- Identification of the activation domain of equine infectious anemia virus rev.
- Platelets from thrombocytopenic ponies acutely infected with equine infectious anemia virus are activated in vivo and hypofunctional.
- Progress in vaccines against AIDS.
- Structural features of the trans-activation response RNA element of equine infectious anemia virus.
- The structural polypeptides of equine infections anemia virus.
- The surface envelope protein gene region of equine infectious anemia virus is not an important determinant of tropism in vitro.