Gluconeogenesis
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Subject Areas on Research
- A sweet spot for the bariatric surgeon.
- Adenovirus-mediated expression of the catalytic subunit of glucose-6-phosphatase in INS-1 cells. Effects on glucose cycling, glucose usage, and insulin secretion.
- Adenovirus-mediated overexpression of liver 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase in gluconeogenic rat hepatoma cells. Paradoxical effect on Fru-2,6-P2 levels.
- Compartmentalized acyl-CoA metabolism in skeletal muscle regulates systemic glucose homeostasis.
- Control of hepatic gluconeogenesis through the transcriptional coactivator PGC-1.
- Cryptococcus neoformans requires a functional glycolytic pathway for disease but not persistence in the host.
- Deletion of CaMKK2 from the liver lowers blood glucose and improves whole-body glucose tolerance in the mouse.
- Dynamic Metabolite Profiling in an Archaeon Connects Transcriptional Regulation to Metabolic Consequences.
- Efficient hepatic glycogen synthesis in refeeding rats requires continued carbon flow through the gluconeogenic pathway.
- FoxO1 deacetylation regulates thyroid hormone-induced transcription of key hepatic gluconeogenic genes.
- Hepatic mTORC1 Opposes Impaired Insulin Action to Control Mitochondrial Metabolism in Obesity.
- New insights into gestational glucose metabolism: lessons learned from 21st century approaches.
- PGC-1beta in the regulation of hepatic glucose and energy metabolism.
- Posttranslational modification of glyceraldehyde-3-phosphate dehydrogenase by S-nitrosylation and subsequent NADH attachment.
- Protein synthesis. The perks of balancing glucose.
- Protein, fat, and carbohydrate requirements during starvation: anaplerosis and cataplerosis.
- Stimulation by alpha-adrenergic agonists of Ca2+ fluxes, mitochondrial oxidation and gluconeogenesis in perfused rat liver.
- The coactivator SRC-1 is an essential coordinator of hepatic glucose production.
- Whole-organism screening for gluconeogenesis identifies activators of fasting metabolism.
- daf-16/FoxO promotes gluconeogenesis and trehalose synthesis during starvation to support survival.
- p38 Mitogen-activated protein kinase plays a stimulatory role in hepatic gluconeogenesis.