Subject Areas on Research
- ApoB-48 and apoB-100 differentially influence the expression of type-III hyperlipoproteinemia in APOE*2 mice.
- Congenital absence of apolipoprotein E and neurological function.
- Treatment of type III hyperlipoproteinemia.
- Type III hyperlipoproteinemia and spontaneous atherosclerosis in mice resulting from gene replacement of mouse Apoe with human Apoe*2.