-
Subject Areas on Research
-
A 13-kilodalton maize mitochondrial protein in E. coli confers sensitivity to Bipolaris maydis toxin.
-
Acylation of naturally occurring and synthetic 1-deoxysphinganines by ceramide synthase. Formation of N-palmitoyl-aminopentol produces a toxic metabolite of hydrolyzed fumonisin, AP1, and a new category of ceramide synthase inhibitor.
-
Alteration of tissue and serum sphinganine to sphingosine ratio: an early biomarker of exposure to fumonisin-containing feeds in pigs.
-
Alterations in reticulo-endothelial functions as produced by the administration of bacterial toxins.
-
Cell cycle progression in G1 and S phases is CCR4 dependent following ionizing radiation or replication stress in Saccharomyces cerevisiae.
-
Chronic toxicity of fumonisins from Fusarium moniliforme culture material (M-1325) to mink.
-
Cross-linking of the cms-T maize mitochondrial pore-forming protein URF13 by N,N'-dicyclohexylcarbodiimide and its effect on URF13 sensitivity to fungal toxins.
-
Dietary fumonisin B1 induces disruption of sphingolipid metabolism in Sprague-Dawley rats: a new mechanism of nephrotoxicity.
-
Effect of nandrolone decanoate administration on recovery from bupivacaine-induced muscle injury.
-
Effects of feeding Fusarium moniliforme culture material, containing known levels of fumonisin B1, on the young broiler chick.
-
Endosymbiosis: the evil within.
-
Evidence for disruption of sphingolipid metabolism as a contributing factor in the toxicity and carcinogenicity of fumonisins.
-
Fluorescence staining of the actin cytoskeleton in living cells with 7-nitrobenz-2-oxa-1,3-diazole-phallacidin.
-
Fumonisin B1 alters sphingolipid metabolism and disrupts the barrier function of endothelial cells in culture.
-
Fumonisin B1 inhibits sphingosine (sphinganine) N-acyltransferase and de novo sphingolipid biosynthesis in cultured neurons in situ.
-
Fumonisin inhibition of de novo sphingolipid biosynthesis and cytotoxicity are correlated in LLC-PK1 cells.
-
Fumonisin toxicity and sphingolipid biosynthesis.
-
Fumonisins and other inhibitors of de novo sphingolipid biosynthesis.
-
Functional expression of the Aspergillus flavus PKS-NRPS hybrid CpaA involved in the biosynthesis of cyclopiazonic acid.
-
Fungal toxins bind to the URF13 protein in maize mitochondria and Escherichia coli.
-
Importance of sphingolipids and inhibitors of sphingolipid metabolism as components of animal diets.
-
In vitro toxicology of fumonisins and the mechanistic implications.
-
Increases in serum sphingosine and sphinganine and decreases in complex sphingolipids in ponies given feed containing fumonisins, mycotoxins produced by Fusarium moniliforme.
-
Inflammatory cytokine gene expression in THP-1 cells exposed to Stachybotrys chartarum and Aspergillus versicolor.
-
Inhibition of sphingolipid biosynthesis by fumonisins. Implications for diseases associated with Fusarium moniliforme.
-
Mutations in the maize mitochondrial T-urf13 gene eliminate sensitivity to a fungal pathotoxin.
-
Mycotoxins and interstitial lung disease.
-
N,N'-dicyclohexylcarbodiimide cross-linking suggests a central core of helices II in oligomers of URF13, the pore-forming T-toxin receptor of cms-T maize mitochondria.
-
Role of dietary sphingolipids and inhibitors of sphingolipid metabolism in cancer and other diseases.
-
Sphingolipid biosynthesis de novo by rat hepatocytes in culture. Ceramide and sphingomyelin are associated with, but not required for, very low density lipoprotein secretion.
-
Sphingolipids as biomarkers of fumonisin exposure and risk of esophageal squamous cell carcinoma in china.
-
Sphingolipids--the enigmatic lipid class: biochemistry, physiology, and pathophysiology.
-
The effect of fumonisin B1 on developing chick embryos: correlation between de novo sphingolipid biosynthesis and gross morphological changes.
-
URF13, a ligand-gated, pore-forming receptor for T-toxin in the inner membrane of cms-T mitochondria.