Myofibrils

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  Subject Areas on Research

  • A mutation in TNNC1-encoded cardiac troponin C, TNNC1-A31S, predisposes to hypertrophic cardiomyopathy and ventricular fibrillation. 
  • Antibodies in sera of patients with late-onset myasthenia gravis recognize the PEVK domain of titin. 
  • Benign vaginal rhabdomyoma: a light and electron microscopic study. 
  • Centronuclear myopathy: histochemistry and electron microscopy. Report of two cases. 
  • Comparative stereology of mouse atria. 
  • Comparative stereology of the lizard and frog myocardium. 
  • Comparative stereology of the mouse and finch left ventricle. 
  • Comparison of myocardial protection with nifedipine and potassium. 
  • Coordinate changes in fast thin filament and Z-line protein expression in the early response to chronic stimulation. 
  • Disruption of myofibrillar energy use: dual mechanisms that may contribute to postischemic dysfunction in stunned myocardium. 
  • Effect of junctional resistance on source-strength in a linear cable. 
  • Effect of oxidative stress on homer scaffolding proteins. 
  • Effects of taxol and Colcemid on myofibrillogenesis. 
  • Field stimulation of cardiac fibers with random spatial structure. 
  • Guidelines for single fiber EMG. 
  • Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma. 
  • Longitudinal variation in muscle protein expression and contraction kinetics of largemouth bass axial muscle. 
  • Membrane properties underlying spontaneous activity of denervated muscle fibres. 
  • Muscle giants: molecular scaffolds in sarcomerogenesis. 
  • Muscle wasting and interleukin-6-induced atrogin-I expression in the cachectic Apc ( Min/+ ) mouse. 
  • Myasthenic syndrome caused by plectinopathy. 
  • Myofiber inclusions. 
  • Pacemaker-induced transient asynchrony suppresses heart failure progression. 
  • Perturbed length-dependent activation in human hypertrophic cardiomyopathy with missense sarcomeric gene mutations. 
  • Plasma creatine kinase activity and exercise-induced muscle damage in older men. 
  • Rapid identification of protein phosphatase 1-binding proteins by mixed peptide sequencing and data base searching. Characterization of a novel holoenzymic form of protein phosphatase 1. 
  • Rapid ventricular pacing of dogs to heart failure: biochemical and physiological studies. 
  • S-Nitrosylation of Sarcomeric Proteins Depresses Myofilament Ca2+)Sensitivity in Intact Cardiomyocytes. 
  • Selective phosphorylation of PKA targets after β-adrenergic receptor stimulation impairs myofilament function in Mybpc3-targeted HCM mouse model. 
  • Sexually dimorphic myofilament function and cardiac troponin I phosphospecies distribution in hypertrophic cardiomyopathy mice. 
  • Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy. 
  • Subsarcolemmal and intermyofibrillar mitochondria play distinct roles in regulating skeletal muscle fatty acid metabolism. 
  • Supramolecular and Liquid Crystalline Contributions to the Assembly of Myofibril. 
  • Sustained correction of motoneuron histopathology following intramuscular delivery of AAV in pompe mice. 
  • The Calcineurin-FoxO-MuRF1 signaling pathway regulates myofibril integrity in cardiomyocytes. 
  • The effect of contractile activity on fibrillation and extrajunctional acetylcholine-sensitivity in rat muscle maintained in organ culture. 
  • The low molecular weight GTPase Rho regulates myofibril formation and organization in neonatal rat ventricular myocytes. Involvement of Rho kinase. 
  • The regulation of skeletal muscle protein turnover during the progression of cancer cachexia in the Apc(Min/+) mouse. 
  • Transgenic mice expressing the myotilin T57I mutation unite the pathology associated with LGMD1A and MFM.