Subject Areas on Research
- CD19 hyperexpression augments Sle1-induced humoral autoimmunity but not clinical nephritis.
- Combined expression of A1 and A20 achieves optimal protection of renal proximal tubular epithelial cells.
- Fas on renal parenchymal cells does not promote autoimmune nephritis in MRL mice.
- Glomerular type 1 angiotensin receptors augment kidney injury and inflammation in murine autoimmune nephritis.
- Hand-assisted laparoscopic nephrectomy for inflammatory renal conditions.
- Hemorrhagic Herpes Simplex Virus Type 1 Nephritis: An Unusual Cause of Acute Allograft Dysfunction.
- Recovery of a human natural antibody against the noncollagenous-1 domain of type IV collagen using humanized models.
- Risk factors for BK polyomavirus nephritis in renal allograft recipients.
- SLCO4C1 transporter eliminates uremic toxins and attenuates hypertension and renal inflammation.
- Selenium-binding protein-1 in smooth muscle cells is downregulated in a rhesus monkey model of chronic allograft nephropathy.
- Systemic autoimmune nephritogenic components induce CSF-1 and TNF-alpha in MRL kidneys.
- TNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α.
- The nephritogenic immune response.
- Transgenic tubular cell expression of class II is insufficient to initiate immune renal injury.
- Transplant approach establishes that kidneys are responsible for serum CSF-1 but require a stimulus in MRL-lpr mice.
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