Osteomalacia

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  Subject Areas on Research

  • A PHEX gene mutation is responsible for adult-onset vitamin D-resistant hypophosphatemic osteomalacia: evidence that the disorder is not a distinct entity from X-linked hypophosphatemic rickets. 
  • Aluminum deposition at the osteoid-bone interface. An epiphenomenon of the osteomalacic state in vitamin D-deficient dogs. 
  • Azotemic renal osteodystrophy. 
  • Burosumab for the Treatment of Tumor-Induced Osteomalacia. 
  • Calcification of entheses associated with X-linked hypophosphatemic osteomalacia. 
  • Drug-induced bone disease. 
  • Equivalency of various methods for estimating osteoid seam width. 
  • Evaluation of a role for 1,25-dihydroxyvitamin D3 in the pathogenesis and treatment of X-linked hypophosphatemic rickets and osteomalacia. 
  • Fibrogenesis imperfecta ossium: MR imaging of the axial and appendicular skeleton and correlation with a unique radiographic appearance. 
  • Following the forgotten phosphorus. 
  • Healing of bone disease in X-linked hypophosphatemic rickets/osteomalacia. Induction and maintenance with phosphorus and calcitriol. 
  • Hypoparathyroidism: a possible cause of osteomalacia . 
  • Hypophosphatemia after intravenous iron therapy: Comprehensive review of clinical findings and recommendations for management. 
  • Hypophosphatemic osteomalacia: association with prostatic carcinoma. 
  • Newer knowledge of vitamin D and its metabolites in health and disease. 
  • Oncogenic osteomalacia caused by phosphaturic mesenchymal tumor of the temporal bone. 
  • Osteomalacia after parathyroidectomy in patients with uremia. 
  • Osteomalacia in chronic renal failure: a syndrome previously reported only with maintenance dialysis. 
  • Postparathyroidectomy hypocalcemia as an accurate indicator of preparathyroidectomy bone histology in the uremic patient. 
  • Reply to: Burosumab for Tumor-Induced Osteomalacia: not Enough of a Good Thing. 
  • Risk Factors for and Effects of Persistent and Severe Hypophosphatemia Following Ferric Carboxymaltose. 
  • Serum 1,25-dihydroxyvitamin D levels in subjects with X-linked hypophosphatemic rickets and osteomalacia. 
  • Spontaneous hypercalcemia in patients undergoing dialysis. Etiologic and therapeutic considerations. 
  • The concurrence of hypoparathyroidism provides new insights to the pathophysiology of X-linked hypophosphatemic rickets. 
  • The efficacy of vitamin D2 and oral phosphorus therapy in X-linked hypophosphatemic rickets and osteomalacia. 
  • The phosphaturic mesenchymal tumor: why is definitive diagnosis and curative surgery often delayed? 
  • Tumor-induced osteomalacia masking primary hyperparathyroidism. 
  • Tumor-induced osteomalacia. 
  • X-Linked hypophosphatemic rickets: a disease often unknown to affected patients. 
  • [Digestive pathology and the osseous system].