Spinal Nerves

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  Subject Areas on Research

  • A feed-forward spinal cord glycinergic neural circuit gates mechanical allodynia. 
  • A peptide c-Jun N-terminal kinase (JNK) inhibitor blocks mechanical allodynia after spinal nerve ligation: respective roles of JNK activation in primary sensory neurons and spinal astrocytes for neuropathic pain development and maintenance. 
  • AXON REGENERATION IN THE HUMAN SPINAL CORD WITH FORMATION OF NEUROMATA. 
  • Anterior intervertebral discectomy and arthrodesis for treatment of low back pain with or without radiculopathy. 
  • Carcinomatous cerebellar degeneration, encephalomyelitis, and sensory neuropathy (radiculitis). Report of a case incorporating both purely degenerative and intense inflammatory changes, with special prominence of the latter in the putamen. 
  • Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain. 
  • ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model. 
  • Effects of ether and thiopental on spinal presynaptic terminals. 
  • Electromyographic evaluation of notalgia paresthetica. 
  • Electromyography as a diagnostic aid in lumbo-sacral nerve root irritation. 
  • Evoked sustained focal potentials and membrane potential of neurons and of unresponsive cells of the spinal cord. 
  • Gene Expression Profiling of Cutaneous Injured and Non-Injured Nociceptors in SNI Animal Model of Neuropathic Pain. 
  • JAK-STAT3 pathway regulates spinal astrocyte proliferation and neuropathic pain maintenance in rats. 
  • Neural control of the urethra. 
  • Pediatric spinal deformities. 
  • Role of the CX3CR1/p38 MAPK pathway in spinal microglia for the development of neuropathic pain following nerve injury-induced cleavage of fractalkine. 
  • Spinal cord neural anatomy in rats examined by in vivo magnetic resonance microscopy. 
  • Sprouting of substance P-expressing primary afferent central terminals and spinal micturition reflex NK1 receptor dependence after spinal cord injury. 
  • Survival Analysis of Occipital Nerve Stimulator Leads Placed under Fluoroscopic Guidance with and without Ultrasonography. 
  • Sustained evoked potential changes of the spinal cord. 
  • The mechanism of the blockade of synaptic transmission in the mammalian spinal cord by diethyl ether and by thiopental. 
  • Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states. 
  • p38 MAPK, microglial signaling, and neuropathic pain. 
  • p38 mitogen-activated protein kinase is activated after a spinal nerve ligation in spinal cord microglia and dorsal root ganglion neurons and contributes to the generation of neuropathic pain.