Spinal Nerves
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Subject Areas on Research
- A feed-forward spinal cord glycinergic neural circuit gates mechanical allodynia.
- A peptide c-Jun N-terminal kinase (JNK) inhibitor blocks mechanical allodynia after spinal nerve ligation: respective roles of JNK activation in primary sensory neurons and spinal astrocytes for neuropathic pain development and maintenance.
- Anterior intervertebral discectomy and arthrodesis for treatment of low back pain with or without radiculopathy.
- Comparison of injectate spread and nerve involvement between retrolaminar and erector spinae plane blocks in the thoracic region: a cadaveric study.
- Different effects of opioid and cannabinoid receptor agonists on C-fiber-induced extracellular signal-regulated kinase activation in dorsal horn neurons in normal and spinal nerve-ligated rats.
- Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain.
- ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model.
- Effects of ether and thiopental on spinal presynaptic terminals.
- Electromyographic evaluation of notalgia paresthetica.
- Electromyography as a diagnostic aid in lumbo-sacral nerve root irritation.
- Evoked sustained focal potentials and membrane potential of neurons and of unresponsive cells of the spinal cord.
- Gene Expression Profiling of Cutaneous Injured and Non-Injured Nociceptors in SNI Animal Model of Neuropathic Pain.
- Influence of injectate volume on paravertebral spread in erector spinae plane block: An endoscopic and anatomical evaluation.
- JAK-STAT3 pathway regulates spinal astrocyte proliferation and neuropathic pain maintenance in rats.
- Large A-fiber activity is required for microglial proliferation and p38 MAPK activation in the spinal cord: different effects of resiniferatoxin and bupivacaine on spinal microglial changes after spared nerve injury.
- Nerve conduction blockade in the sciatic nerve prevents but does not reverse the activation of p38 mitogen-activated protein kinase in spinal microglia in the rat spared nerve injury model.
- Neural control of the urethra.
- Notalgia paresthetica.
- Pediatric spinal deformities.
- Role of computed tomography in the radiological evaluation of painful radiculopathy after negative myelography: foraminal neural entrapment.
- Role of the CX3CR1/p38 MAPK pathway in spinal microglia for the development of neuropathic pain following nerve injury-induced cleavage of fractalkine.
- Spinal cord neural anatomy in rats examined by in vivo magnetic resonance microscopy.
- Sprouting of substance P-expressing primary afferent central terminals and spinal micturition reflex NK1 receptor dependence after spinal cord injury.
- Survival Analysis of Occipital Nerve Stimulator Leads Placed under Fluoroscopic Guidance with and without Ultrasonography.
- Sustained evoked potential changes of the spinal cord.
- The effects of stimulating the dorsal columns of man.
- The mechanism of the blockade of synaptic transmission in the mammalian spinal cord by diethyl ether and by thiopental.
- Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states.
- Timing of sacral neurostimulation is important for increasing bladder capacity in the anesthetized rat.
- p38 MAPK, microglial signaling, and neuropathic pain.
- p38 mitogen-activated protein kinase is activated after a spinal nerve ligation in spinal cord microglia and dorsal root ganglion neurons and contributes to the generation of neuropathic pain.