Receptors, HIV
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Subject Areas on Research
- AIDS. Do antibodies enhance the infection of cells by HIV?
- CD4-independent binding of HIV-1 to the B lymphocyte receptor CR2 (CD21) in the presence of complement and antibody.
- CX3CR1 tyrosine sulfation enhances fractalkine-induced cell adhesion.
- Chemokines, neuronal-glial interactions, and central processing of neuropathic pain.
- Envelope V3 amino acid sequence predicts HIV-1 phenotype (co-receptor usage and tropism for macrophages).
- Global panel of HIV-1 Env reference strains for standardized assessments of vaccine-elicited neutralizing antibodies.
- HIV fusion and its inhibition.
- HIV type 1 from a patient with baseline resistance to CCR5 antagonists uses drug-bound receptor for entry.
- HIV-1 GP120-mediated immune suppression and lymphocyte destruction in the absence of viral infection.
- HIV-1 neutralization by chimeric CD4-CG10 polypeptides fused to human IgG1.
- HIV-1 resistance to CCR5 antagonists associated with highly efficient use of CCR5 and altered tropism on primary CD4+ T cells.
- HIV-1-induced cell fusion is mediated by multiple regions within both the viral envelope and the CCR-5 co-receptor.
- Identification of a novel cell attachment domain in the HIV-1 Tat protein and its 90-kDa cell surface binding protein.
- Immunologic and virologic analyses of an acutely HIV type 1-infected patient with extremely rapid disease progression.
- Molecular mechanism of desensitization of the chemokine receptor CCR-5: receptor signaling and internalization are dissociable from its role as an HIV-1 co-receptor.
- Multiple blocks to human immunodeficiency virus type 1 replication in rodent cells.
- Murine CXCR-4 is a functional coreceptor for T-cell-tropic and dual-tropic strains of human immunodeficiency virus type 1.
- Mutational analysis of the fractalkine chemokine domain. Basic amino acid residues differentially contribute to CX3CR1 binding, signaling, and cell adhesion.
- Neutralizing antibodies to an immunodominant envelope sequence do not prevent gp120 binding to CD4.
- Noncytolytic CD8 T cell-mediated suppression of HIV replication.
- Origin and evolution of HIV-1 in breast milk determined by single-genome amplification and sequencing.
- Postexposure immunoprophylaxis of primary isolates by an antibody to HIV receptor complex.
- Primary infection by a human immunodeficiency virus with atypical coreceptor tropism.
- Structural mechanism of trimeric HIV-1 envelope glycoprotein activation.
- The ability of HIV type 1 to use CCR-3 as a coreceptor is controlled by envelope V1/V2 sequences acting in conjunction with a CCR-5 tropic V3 loop.
- The phenotype of human placental macrophages and its variation with gestational age.
- Transmission of Multiple HIV-1 Subtype C Transmitted/founder Viruses into the Same Recipients Was not Determined by Modest Phenotypic Differences.