NAD(P)H Dehydrogenase (Quinone)
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Subject Areas on Research
- Autophagy protects against aminochrome-induced cell death in substantia nigra-derived cell line.
- Comparative analysis of the zeta-crystallin/quinone reductase gene in guinea pig and mouse.
- Discovery of novel targets of quinoline drugs in the human purine binding proteome.
- Evidence for independent recruitment of zeta-crystallin/quinone reductase (CRYZ) as a crystallin in camelids and hystricomorph rodents.
- Genetic susceptibility to benzene-induced toxicity: role of NADPH: quinone oxidoreductase-1.
- Mitochondrial generation of reactive oxygen species after brain ischemia in the rat.
- Modelling the effects of age-related mtDNA mutation accumulation; complex I deficiency, superoxide and cell death.
- NAD(P)H quinone oxidoreductase 1 is essential for ozone-induced oxidative stress in mice and humans.
- NAD(P)H quinone oxidoreductase 1 regulates neutrophil elastase-induced mucous cell metaplasia.
- NADPH:quinone oxidoreductase 1 regulates host susceptibility to ozone via isoprostane generation.
- Organization of the human zeta-crystallin/quinone reductase gene (CRYZ).
- Overexpression of VMAT-2 and DT-diaphorase protects substantia nigra-derived cells against aminochrome neurotoxicity.
- Quinone oxidoreductase (NQO1) gene polymorphism may not confer a susceptibility to mood disorders.
- RETRACTED: NAD(P)H:quinone oxidoreductase 1 protects lungs from oxidant-induced emphysema in mice.
- Regulation of MUC5AC expression by NAD(P)H:quinone oxidoreductase 1.
- Xenobiotic-Metabolizing gene polymorphisms and ovarian cancer risk.
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Keywords of People
- Berchuck, Andrew, James M. Ingram Professor of Gynecologic Oncology, Obstetrics and Gynecology, Gynecologic Oncology