Genes, Helminth
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Subject Areas on Research
- Alteration of Caenorhabditis elegans gene expression by targeted transformation.
- An essential role for DYF-11/MIP-T3 in assembling functional intraflagellar transport complexes.
- DEPDC1/LET-99 participates in an evolutionarily conserved pathway for anti-tubulin drug-induced apoptosis.
- GATA transcription factor required for immunity to bacterial and fungal pathogens.
- Identification of regulators of germ stem cell enwrapment by its niche in C. elegans.
- Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.
- MIG-10 (lamellipodin) has netrin-independent functions and is a FOS-1A transcriptional target during anchor cell invasion in C. elegans.
- Neural regulation of immunity: role of NPR-1 in pathogen avoidance and regulation of innate immunity.
- Neuronal GPCR controls innate immunity by regulating noncanonical unfolded protein response genes.
- Nucleolar proteins suppress Caenorhabditis elegans innate immunity by inhibiting p53/CEP-1.
- Overcoming redundancy: an RNAi enhancer screen for morphogenesis genes in Caenorhabditis elegans.
- Pairing of competitive and topologically distinct regulatory modules enhances patterned gene expression.
- Patterns of flanking sequence conservation and a characteristic upstream motif for microRNA gene identification.
- Pol II docking and pausing at growth and stress genes in C. elegans.
- RNA Pol II accumulates at promoters of growth genes during developmental arrest.
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Keywords of People
- Davis, Erica Ellen, Associate Professor of Pediatrics, Cell Biology