Subject Areas on Research
- Allospecific CD4(+) effector memory T cells do not induce graft-versus-host disease in mice.
- Anergy during pregnancy.
- CD19-regulated signaling thresholds control peripheral tolerance and autoantibody production in B lymphocytes.
- CD95 (Fas)-dependent elimination of self-reactive B cells upon interaction with CD4+ T cells.
- Cutting edge: hierarchy of chemokine receptor and TCR signals regulating T cell migration and proliferation.
- Delayed-type hypersensitivity testing in Tanzanian adults with HIV infection.
- HIV-1 gp120 induces anergy in naive T lymphocytes through CD4-independent protein kinase-A-mediated signaling.
- Improving Mycobacterium bovis bacillus Calmette-Guèrin as a vaccine delivery vector for viral antigens by incorporation of glycolipid activators of NKT cells.
- Inhibition of NF-kappa B and oxidative pathways in human dendritic cells by antioxidative vitamins generates regulatory T cells.
- Lack of galectin-1 or galectin-3 alters B cell deletion and anergy in an autoantibody transgene model.
- Metabolic reprogramming is required for antibody production that is suppressed in anergic but exaggerated in chronically BAFF-exposed B cells.
- Multifunctional regulators of cell growth are differentially expressed in anergic murine B cells.
- Redemption of autoreactive B cells.
- Repression of B7.2 on self-reactive B cells is essential to prevent proliferation and allow Fas-mediated deletion by CD4(+) T cells.
- The road to tolerance: renal transplant tolerance induction in nonhuman primate studies and clinical trials.
- Tuberous sclerosis 1 promotes invariant NKT cell anergy and inhibits invariant NKT cell-mediated antitumor immunity.
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