Sodium Compounds
-
Subject Areas on Research
- Apoptosis and P53 induction in human lung fibroblasts exposed to chromium (VI): effect of ascorbate and tocopherol.
- Apoptosis is the mode of cell death caused by carcinogenic chromium.
- Cellular shear stiffness reflects progression of arsenic-induced transformation during G1.
- Chromium (VI) activates ataxia telangiectasia mutated (ATM) protein. Requirement of ATM for both apoptosis and recovery from terminal growth arrest.
- Cyclosporin A inhibits chromium(VI)-induced apoptosis and mitochondrial cytochrome c release and restores clonogenic survival in CHO cells.
- Deficiencies in mitochondrial dynamics sensitize Caenorhabditis elegans to arsenite and other mitochondrial toxicants by reducing mitochondrial adaptability.
- Differential impact of ionic and coordinate covalent chromium (Cr)-DNA binding on DNA replication.
- Effects of perchlorate on the molecules of excitation-contraction coupling of skeletal and cardiac muscle.
- Induction of internucleosomal DNA fragmentation by carcinogenic chromate: relationship to DNA damage, genotoxicity, and inhibition of macromolecular synthesis.
- Mechanism of apoptosis and determination of cellular fate in chromium(VI)-exposed populations of telomerase-immortalized human fibroblasts.
- Perchlorate reduction by autotrophic bacteria in the presence of zero-valent iron.
- Rapid measurement of total body water to facilitate clinical decision making in hospitalized elderly patients.
- Reduced arsenic clearance and increased toxicity in aquaglyceroporin-9-null mice.
- Resistance to apoptosis, increased growth potential, and altered gene expression in cells that survived genotoxic hexavalent chromium [Cr(VI)] exposure.
- Sodium-iodide symporter (NIS)-mediated accumulation of [(211)At]astatide in NIS-transfected human cancer cells.
- Transcriptional inhibition by carcinogenic chromate: relationship to DNA damage.
- Use of sodium chromate Cr51 in diagnosing childhood idiopathic pulmonary hemosiderosis.