Epstein-Barr Virus Nuclear Antigens
Subject Areas on Research
- An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cells.
- An altered immune response to Epstein-Barr nuclear antigen 1 in pediatric systemic lupus erythematosus.
- Combined assessment of Epstein-Barr virus viral capsid antigen and Epstein-Barr virus nuclear antigen-1 serology for post-transplant lymphoproliferative disorder risk stratification in adult solid organ transplant recipients.
- Early events in lupus humoral autoimmunity suggest initiation through molecular mimicry.
- Enhanced outgrowth of EBV-transformed chronic lymphocytic leukemia B cells mediated by coculture with macrophage feeder cells.
- Epstein Barr virus nuclear antigen 1 (EBNA-1) peptides recognized by adult multiple sclerosis patient sera induce neurologic symptoms in a murine model.
- Epstein-Barr Virus Genomes Reveal Population Structure and Type 1 Association with Endemic Burkitt Lymphoma.
- Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection.
- Epstein-Barr virus-associated smooth muscle tumors are distinctive mesenchymal tumors reflecting multiple infection events: a clinicopathologic and molecular analysis of 29 tumors from 19 patients.
- Infectious mononucleosis patients temporarily recognize a unique, cross-reactive epitope of Epstein-Barr virus nuclear antigen-1.
Keywords of People
- Luftig, Micah Alan, Professor of Molecular Genetics and Microbiology, Cell Biology