Subject Areas on Research
- An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cells.
- An altered immune response to Epstein-Barr nuclear antigen 1 in pediatric systemic lupus erythematosus.
- Current and past Epstein-Barr virus infection in risk of initial CNS demyelination.
- Early events in lupus humoral autoimmunity suggest initiation through molecular mimicry.
- Enhanced outgrowth of EBV-transformed chronic lymphocytic leukemia B cells mediated by coculture with macrophage feeder cells.
- Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection.
- Epstein-Barr virus-associated smooth muscle tumors are distinctive mesenchymal tumors reflecting multiple infection events: a clinicopathologic and molecular analysis of 29 tumors from 19 patients.
- Infectious mononucleosis patients temporarily recognize a unique, cross-reactive epitope of Epstein-Barr virus nuclear antigen-1.
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