Macrophage Inflammatory Proteins
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Subject Areas on Research
- CD8 positive T cells influence antigen-specific immune responses through the expression of chemokines.
- Chemokine production by G protein-coupled receptor activation in a human mast cell line: roles of extracellular signal-regulated kinase and NFAT.
- Differential regulation of beta-chemokines in children with Plasmodium falciparum malaria.
- Endotoxin and NO induce MIP-1gamma gene transcription in ANA-1 murine macrophages.
- Enhanced expression of cytokines and chemokines by blood monocytes to in vitro lipopolysaccharide stimulation are associated with hostility and severity of depressive symptoms in healthy women.
- Evidence that antibody-mediated neutralization of human immunodeficiency virus type 1 by sera from infected individuals is independent of coreceptor usage.
- HIV-1 Nef protein: An invitation to a kill.
- Herpesvirus saimiri transformation of HIV type 1 suppressive CD8+ lymphocytes from an HIV type 1-infected asymptomatic individual.
- Human placental cytotrophoblasts attract monocytes and CD56(bright) natural killer cells via the actions of monocyte inflammatory protein 1alpha.
- Identification of a glycosaminoglycan-binding site in chemokine macrophage inflammatory protein-1alpha.
- In vivo cytokine-associated responses to biomaterials.
- Intracellular adhesion molecule-1 modulates beta-chemokines and directly costimulates T cells in vivo.
- Neutralizing antibody responses to human immunodeficiency virus type 1 in primary infection and long-term-nonprogressive infection.
- Pathogenesis of pneumococcal pneumonia in cyclophosphamide-induced leukopenia in mice.
- Reduced GRK2 level in T cells potentiates chemotaxis and signaling in response to CCL4.
- Regulation of I-309 gene expression in human monocytes by endogenous interleukin-1.
- Requirement of MIP-1 alpha for an inflammatory response to viral infection.
- Serum cytokine profiles in relapsing polychondritis suggest monocyte/macrophage activation.
- Structure and function of the glycosaminoglycan binding site of chemokine macrophage-inflammatory protein-1 beta.
- Ultrafine carbon black particles enhance respiratory syncytial virus-induced airway reactivity, pulmonary inflammation, and chemokine expression.
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Keywords of People
- Krangel, Michael S., George Barth Geller Distinguished Professor of Immunology, Immunology