Hindlimb Suspension

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  Subject Areas on Research

  • A model of muscle atrophy using cast immobilization in mice. 
  • Alterations in inorganic phosphate in mouse hindlimb muscles during limb disuse. 
  • Cortical-amygdalar circuit dysfunction in a genetic mouse model of serotonin deficiency. 
  • Dynamically Timed Stimulation of Corticolimbic Circuitry Activates a Stress-Compensatory Pathway. 
  • Effects of spaceflight on murine skeletal muscle gene expression. 
  • Emergence of anxiety-like behaviours in depressive-like Cpe(fat/fat) mice. 
  • Involvement of AMPA receptors in the antidepressant-like effects of lithium in the mouse tail suspension test and forced swim test. 
  • Noninvasive monitoring of muscle damage during reloading following limb disuse. 
  • PGC1-α over-expression prevents metabolic alterations and soleus muscle atrophy in hindlimb unloaded mice. 
  • Skeletal muscle mass recovery from atrophy in IL-6 knockout mice. 
  • The role of alterations in mitochondrial dynamics and PGC-1α over-expression in fast muscle atrophy following hindlimb unloading. 
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  Keywords of People

  • Nicolelis, Miguel Angelo L., Duke School of Medicine Distinguished Professor in Neuroscience, Neurobiology