Hindlimb Suspension
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Subject Areas on Research
- A model of muscle atrophy using cast immobilization in mice.
- Alterations in inorganic phosphate in mouse hindlimb muscles during limb disuse.
- Cortical-amygdalar circuit dysfunction in a genetic mouse model of serotonin deficiency.
- Dynamically Timed Stimulation of Corticolimbic Circuitry Activates a Stress-Compensatory Pathway.
- Effects of spaceflight on murine skeletal muscle gene expression.
- Emergence of anxiety-like behaviours in depressive-like Cpe(fat/fat) mice.
- Involvement of AMPA receptors in the antidepressant-like effects of lithium in the mouse tail suspension test and forced swim test.
- Noninvasive monitoring of muscle damage during reloading following limb disuse.
- PGC1-α over-expression prevents metabolic alterations and soleus muscle atrophy in hindlimb unloaded mice.
- Skeletal muscle mass recovery from atrophy in IL-6 knockout mice.
- The role of alterations in mitochondrial dynamics and PGC-1α over-expression in fast muscle atrophy following hindlimb unloading.
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Keywords of People
- Nicolelis, Miguel Angelo L., Duke School of Medicine Distinguished Professor in Neuroscience, Neurobiology