CCAAT-Enhancer-Binding Protein-beta
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Subject Areas on Research
- C/EBPβ controls exercise-induced cardiac growth and protects against pathological cardiac remodeling.
- CCAAT/enhancer-binding protein beta deletion reduces adiposity, hepatic steatosis, and diabetes in Lepr(db/db) mice.
- Cyclic regulation of transcription factor C/EBP beta in human endometrium.
- Glutamine-mediated dual regulation of heat shock transcription factor-1 activation and expression.
- Growth hormone regulates phosphorylation and function of CCAAT/enhancer-binding protein beta by modulating Akt and glycogen synthase kinase-3.
- Human DNA methylation signatures differentiate persistent from resolving MRSA bacteremia.
- Identification of a tissue-specific, C/EBPβ-dependent pathway of differentiation for murine peritoneal macrophages.
- Malignant breast epithelial cells stimulate aromatase expression via promoter II in human adipose fibroblasts: an epithelial-stromal interaction in breast tumors mediated by CCAAT/enhancer binding protein beta.
- Mice with a deletion in the gene for CCAAT/enhancer-binding protein beta are protected against diet-induced obesity.
- Molecular regulation of constitutive expression of interleukin-8 in human pancreatic adenocarcinoma.
- Mouse adenovirus type 1 infection of adipose tissue.
- NF-kappaB factors are essential, but not the switch, for pathogen-related induction of the bovine beta-defensin 5-encoding gene in mammary epithelial cells.
- Nuclear factor erythroid-derived factor 2-related factor 2 regulates transcription of CCAAT/enhancer-binding protein β during adipogenesis.
- PARP-1 Controls the Adipogenic Transcriptional Program by PARylating C/EBPβ and Modulating Its Transcriptional Activity.