WT1 Proteins
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Subject Areas on Research
- A novel missense mutation of Wilms' Tumor 1 causes autosomal dominant FSGS.
- COQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness.
- Clinical outcome and gene- and microRNA-expression profiling according to the Wilms tumor 1 (WT1) single nucleotide polymorphism rs16754 in adult de novo cytogenetically normal acute myeloid leukemia: a Cancer and Leukemia Group B study.
- Colocalization of WT1 and cell proliferation reveals conserved mechanisms in temperature-dependent sex determination.
- Dysregulation of WTI (-KTS) is Associated with the Kidney-Specific Effects of the LMX1B R246Q Mutation.
- Estrogen represses SOX9 during sex determination in the red-eared slider turtle Trachemys scripta.
- HIV-1 Nef induces dedifferentiation of podocytes in vivo: a characteristic feature of HIVAN.
- HIV-1 induces renal epithelial dedifferentiation in a transgenic model of HIV-associated nephropathy.
- Immunotherapeutic targeting of Wilms' tumor protein.
- Induction of Wilms' tumor protein (WT1)-specific antitumor immunity using a truncated WT1-expressing adenovirus vaccine.
- Mutational analysis of NPHS2 and WT1 in frequently relapsing and steroid-dependent nephrotic syndrome.
- Mutations in PLCE1 are a major cause of isolated diffuse mesangial sclerosis (IDMS).
- Nephrotic syndrome in the first year of life: two thirds of cases are caused by mutations in 4 genes (NPHS1, NPHS2, WT1, and LAMB2).
- Restricted expression of WT1 messenger ribonucleic acid in immature ovarian follicles: uniformity in mammalian and avian species and maintenance during reproductive senescence.
- WT1 and DAX-1 inhibit aromatase P450 expression in human endometrial and endometriotic stromal cells.
- WT1 and DAX-1 regulate SF-1-mediated human P450arom gene expression in gonadal cells.
- WT1 vaccination in acute myeloid leukemia: new methods of implementing adoptive immunotherapy.