Pulmonary Surfactant-Associated Protein A
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Subject Areas on Research
- Altered surfactant function and structure in SP-A gene targeted mice.
- Bombesin-like peptides and receptors in normal fetal baboon lung: roles in lung growth and maturation.
- Cryptococcus neoformans is resistant to surfactant protein A mediated host defense mechanisms.
- Embryonic mouse lung epithelial progenitor cells co-express immunohistochemical markers of diverse mature cell lineages.
- Eosinophil-associated lung diseases. A cry for surfactant proteins A and D help?
- Genetic Variation in Surfactant Protein-A2 Delays Resolution of Eosinophilia in Asthma.
- Genetic variation in surfactant protein-A2 alters responses to ozone.
- Identification and Quantitation of Coding Variants and Isoforms of Pulmonary Surfactant Protein A.
- Immunomodulatory roles of surfactant proteins A and D: implications in lung disease.
- Incorporation of biotinylated SP-A into rat lung surfactant layer, type II cells, and clara cells.
- Lung effector memory and activated CD4+ T cells display enhanced proliferation in surfactant protein A-deficient mice during allergen-mediated inflammation.
- Mast cell TNF receptors regulate responses to Mycoplasma pneumoniae in surfactant protein A (SP-A)-/- mice.
- Nitric oxide mediates relative airway hyporesponsiveness to lipopolysaccharide in surfactant protein A-deficient mice.
- Novel role for surfactant protein A in gastrointestinal graft-versus-host disease.
- Obese asthmatic patients have decreased surfactant protein A levels: Mechanisms and implications.
- Proliferation and differentiation defects during lung development in corticotropin-releasing hormone-deficient mice.
- Pseudomonas aeruginosa elastase degrades surfactant proteins A and D.
- Review: Collectins link innate and adaptive immunity in allergic airway disease.
- SP-A preserves airway homeostasis during Mycoplasma pneumoniae infection in mice.
- Surfactant apoprotein in adult rat lung compartments is increased by dexamethasone.
- Surfactant protein A integrates activation signal strength to differentially modulate T cell proliferation.
- Surfactant protein A is defective in abrogating inflammation in asthma.
- Surfactant protein A modulates induction of regulatory T cells via TGF-β.
- Surfactant protein D decreases pollen-induced IgE-dependent mast cell degranulation.
- Surfactant protein-A inhibits mycoplasma-induced dendritic cell maturation through regulation of HMGB-1 cytokine activity.
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Keywords of People
- Cianciolo, George James, Associate Professor Emeritus of Pathology, Pathology