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Pulmonary Surfactant-Associated Protein C
Subject Areas on Research
Airway injury in lung disease pathophysiology: selective depletion of airway stem and progenitor cell pools potentiates lung inflammation and alveolar dysfunction.
Hyperoxia impairs postnatal alveolar epithelial development via NADPH oxidase in newborn mice.
Molecular phenotype of airway side population cells.
Nonspecific interstitial pneumonia, alveolar proteinosis, and abnormal proprotein trafficking resulting from a spontaneous mutation in the surfactant protein C gene.
