Pulmonary Surfactant-Associated Protein C
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Subject Areas on Research
- Airway injury in lung disease pathophysiology: selective depletion of airway stem and progenitor cell pools potentiates lung inflammation and alveolar dysfunction.
- Evidence for type II cells as cells of origin of K-Ras-induced distal lung adenocarcinoma.
- Fetal alveolar epithelial cells contain [D-Ala(2)]-deltorphin I-like immunoreactivity: delta- and mu-opiate receptors mediate opposite effects in developing lung.
- Hyperoxia impairs postnatal alveolar epithelial development via NADPH oxidase in newborn mice.
- Molecular phenotype of airway side population cells.
- Nmyc plays an essential role during lung development as a dosage-sensitive regulator of progenitor cell proliferation and differentiation.
- Nonspecific interstitial pneumonia, alveolar proteinosis, and abnormal proprotein trafficking resulting from a spontaneous mutation in the surfactant protein C gene.
- Resequencing Study Confirms That Host Defense and Cell Senescence Gene Variants Contribute to the Risk of Idiopathic Pulmonary Fibrosis.
- Telomere dysfunction causes alveolar stem cell failure.
- Transgenic over-expression of the microRNA miR-17-92 cluster promotes proliferation and inhibits differentiation of lung epithelial progenitor cells.