Pseudomonas syringae
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Subject Areas on Research
- A bacterial tyrosine phosphatase inhibits plant pattern recognition receptor activation.
- A bacterial virulence protein suppresses host innate immunity to cause plant disease.
- A critical role of STAYGREEN/Mendel's I locus in controlling disease symptom development during Pseudomonas syringae pv tomato infection of Arabidopsis.
- A critical role of two positively charged amino acids in the Jas motif of Arabidopsis JAZ proteins in mediating coronatine- and jasmonoyl isoleucine-dependent interactions with the COI1 F-box protein.
- A family of conserved bacterial effectors inhibits salicylic acid-mediated basal immunity and promotes disease necrosis in plants.
- A genetic screen reveals Arabidopsis stomatal and/or apoplastic defenses against Pseudomonas syringae pv. tomato DC3000.
- A kiss of death--proteasome-mediated membrane fusion and programmed cell death in plant defense against bacterial infection.
- A noncanonical role for the CKI-RB-E2F cell-cycle signaling pathway in plant effector-triggered immunity.
- A prominent role of the flagellin receptor FLAGELLIN-SENSING2 in mediating stomatal response to Pseudomonas syringae pv tomato DC3000 in Arabidopsis.
- Apoplastic peroxidases are required for salicylic acid-mediated defense against Pseudomonas syringae.
- Arabidopsis BRCA2 and RAD51 proteins are specifically involved in defense gene transcription during plant immune responses.
- Arabidopsis calcium-dependent protein kinase 3 regulates actin cytoskeleton organization and immunity.
- Bacteria establish an aqueous living space in plants crucial for virulence.
- Bacterial effector activates jasmonate signaling by directly targeting JAZ transcriptional repressors.
- Coronatine promotes Pseudomonas syringae virulence in plants by activating a signaling cascade that inhibits salicylic acid accumulation.
- DNA repair proteins are directly involved in regulation of gene expression during plant immune response.
- De novo assembly using low-coverage short read sequence data from the rice pathogen Pseudomonas syringae pv. oryzae.
- Diverse mechanisms of resistance to Pseudomonas syringae in a thousand natural accessions of Arabidopsis thaliana.
- Dual impact of elevated temperature on plant defence and bacterial virulence in Arabidopsis.
- Effector-triggered immunity blocks pathogen degradation of an immunity-associated vesicle traffic regulator in Arabidopsis.
- Functional characterization of a Nudix hydrolase AtNUDX8 upon pathogen attack indicates a positive role in plant immune responses.
- Genome-wide transcriptional analysis of the Arabidopsis thaliana interaction with the plant pathogen Pseudomonas syringae pv. tomato DC3000 and the human pathogen Escherichia coli O157:H7.
- Host target modification as a strategy to counter pathogen hijacking of the jasmonate hormone receptor.
- Induction and suppression of PEN3 focal accumulation during Pseudomonas syringae pv. tomato DC3000 infection of Arabidopsis.
- Localization of DIR1 at the tissue, cellular and subcellular levels during Systemic Acquired Resistance in Arabidopsis using DIR1:GUS and DIR1:EGFP reporters.
- Making sense of hormone crosstalk during plant
- Overexpression of Arabidopsis MAP kinase kinase 7 leads to activation of plant basal and systemic acquired resistance.
- Pathogenic Bacteria Target Plant Plasmodesmata to Colonize and Invade Surrounding Tissues.
- Pattern-recognition receptors are required for NLR-mediated plant immunity.
- Plant Immunity Requires Conformational Changes of NPR1 via S-Nitrosylation and Thioredoxins
- Plant stomata function in innate immunity against bacterial invasion.
- Pseudomonas syringae Effector Avirulence Protein E Localizes to the Host Plasma Membrane and Down-Regulates the Expression of the NONRACE-SPECIFIC DISEASE RESISTANCE1/HARPIN-INDUCED1-LIKE13 Gene Required for Antibacterial Immunity in Arabidopsis.
- Pseudomonas syringae infection assays in Arabidopsis.
- Pseudomonas syringae pv. tomato DC3000: a model pathogen for probing disease susceptibility and hormone signaling in plants.
- Pseudomonas syringae: what it takes to be a pathogen.
- Quantitative in situ assay of salicylic acid in tobacco leaves using a genetically modified biosensor strain of Acinetobacter sp. ADP1.
- Redox rhythm reinforces the circadian clock to gate immune response
- Regulation of plant arginase by wounding, jasmonate, and the phytotoxin coronatine.
- Role of plant stomata in bacterial invasion.
- Salicylic acid biosynthesis is enhanced and contributes to increased biotrophic pathogen resistance in Arabidopsis hybrids.
- Subcellular Localization of Pseudomonas syringae pv. tomato Effector Proteins in Plants.
- Subcellular localization and functional analysis of the Arabidopsis GTPase RabE.
- Suppression of host defense in compatible plant-Pseudomonas syringae interactions.
- The Erwinia amylovora avrRpt2EA gene contributes to virulence on pear and AvrRpt2EA is recognized by Arabidopsis RPS2 when expressed in pseudomonas syringae.
- The Pseudomonas syringae Type III Effector HopG1 Induces Actin Remodeling to Promote Symptom Development and Susceptibility during Infection.
- The Pseudomonas syringae type III effector tyrosine phosphatase HopAO1 suppresses innate immunity in Arabidopsis thaliana.
- The bacterial effector HopM1 suppresses PAMP-triggered oxidative burst and stomatal immunity.
- Transcriptome landscape of a bacterial pathogen under plant immunity.
- Translational Regulation of Metabolic Dynamics during Effector-Triggered Immunity.
- Use of dominant-negative HrpA mutants to dissect Hrp pilus assembly and type III secretion in Pseudomonas syringae pv. tomato.
- Virulence systems of Pseudomonas syringae pv. tomato promote bacterial speck disease in tomato by targeting the jasmonate signaling pathway.