Pseudomonas syringae
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Subject Areas on Research
- A kiss of death--proteasome-mediated membrane fusion and programmed cell death in plant defense against bacterial infection.
- A noncanonical role for the CKI-RB-E2F cell-cycle signaling pathway in plant effector-triggered immunity.
- Apoplastic peroxidases are required for salicylic acid-mediated defense against Pseudomonas syringae.
- Arabidopsis BRCA2 and RAD51 proteins are specifically involved in defense gene transcription during plant immune responses.
- Coronatine promotes Pseudomonas syringae virulence in plants by activating a signaling cascade that inhibits salicylic acid accumulation.
- DNA repair proteins are directly involved in regulation of gene expression during plant immune response.
- De novo assembly using low-coverage short read sequence data from the rice pathogen Pseudomonas syringae pv. oryzae.
- Functional characterization of a Nudix hydrolase AtNUDX8 upon pathogen attack indicates a positive role in plant immune responses.
- Making sense of hormone crosstalk during plant immune responses.
- Overexpression of Arabidopsis MAP kinase kinase 7 leads to activation of plant basal and systemic acquired resistance.
- Plant immunity requires conformational changes [corrected] of NPR1 via S-nitrosylation and thioredoxins.
- Pseudomonas syringae Effector Avirulence Protein E Localizes to the Host Plasma Membrane and Down-Regulates the Expression of the NONRACE-SPECIFIC DISEASE RESISTANCE1/HARPIN-INDUCED1-LIKE13 Gene Required for Antibacterial Immunity in Arabidopsis.
- Redox rhythm reinforces the circadian clock to gate immune response.
- Salicylic acid biosynthesis is enhanced and contributes to increased biotrophic pathogen resistance in Arabidopsis hybrids.