Tumor Necrosis Factors

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  Subject Areas on Research

  • A Novel Analytic Technique to Measure Associations Between Circulating Biomarkers and Physical Performance Across the Adult Life Span. 
  • Activated B cells modified by electroporation of multiple mRNAs encoding immune stimulatory molecules are comparable to mature dendritic cells in inducing in vitro antigen-specific T-cell responses. 
  • Construction and preclinical characterization of Fc-mGITRL for the immunotherapy of cancer. 
  • Enhancement of anti-tumor immunity through local modulation of CTLA-4 and GITR by dendritic cells. 
  • Functional polymorphisms in the promoter regions of the FAS and FAS ligand genes and risk of bladder cancer in south China: a case-control analysis. 
  • Gender and estrogen supplementation increases severity of experimental choroidal neovascularization. 
  • Heme induces programmed necrosis on macrophages through autocrine TNF and ROS production. 
  • Maternal alloantigens promote the development of tolerogenic fetal regulatory T cells in utero. 
  • Nuclear factor-kappaB mediates Kupffer cell apoptosis through transcriptional activation of Fas/FasL. 
  • Polymorphisms of the FAS and FAS ligand genes associated with risk of cutaneous malignant melanoma. 
  • Precision nephrology identified tumor necrosis factor activation variability in minimal change disease and focal segmental glomerulosclerosis. 
  • RNPS1 inhibits excessive tumor necrosis factor/tumor necrosis factor receptor signaling to support hematopoiesis in mice. 
  • Recombinant HMGB1 with cytokine-stimulating activity. 
  • Response of brain tissue to chronically implanted neural electrodes. 
  • Spatiotemporal programming of a simple inflammatory process. 
  • Studies on the interaction between TWEAK and the death receptor WSL-1/TRAMP (DR3). 
  • TWEAK/Fn14 signaling is required for liver regeneration after partial hepatectomy in mice. 
  • The "cytokine profile": a code for sepsis. 
  • The role of p65 NF-kappaB/RelA in pancreatitis-induced Kupffer cell apoptosis. 
  • Transgenic expression of the viral FLIP MC159 causes lpr/gld-like lymphoproliferation and autoimmunity. 
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  Keywords of People

  • Choi, Steven Sok, Associate Professor of Medicine, Medicine, Gastroenterology