Receptors, Lysosphingolipid
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Subject Areas on Research
- CD4 T follicular helper cell dynamics during SIV infection.
- High-fat/high-cholesterol diet promotes a S1P receptor-mediated antiapoptotic activity for VLDL.
- Human neural progenitors express functional lysophospholipid receptors that regulate cell growth and morphology.
- Immunosurveillance by hematopoietic progenitor cells trafficking through blood, lymph, and peripheral tissues.
- Meta-analysis of rare and common exome chip variants identifies S1PR4 and other loci influencing blood cell traits.
- Molecular characterization of circulating plasma cells in patients with active systemic lupus erythematosus.
- Novel sphingosine-1-phosphate receptor modulator KRP203 combined with locally delivered regulatory T cells induces permanent acceptance of pancreatic islet allografts.
- P2X7 receptor agonists pre- and postcondition the heart against ischemia-reperfusion injury by opening pannexin-1/P2X₇ channels.
- PTEN as an effector in the signaling of antimigratory G protein-coupled receptor.
- Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes.
- Pharmacological modulation of peripheral T and B lymphocytes by a selective sphingosine 1-phosphate receptor-1 modulator.
- Phenotypic regulation of the sphingosine 1-phosphate receptor miles apart by G protein-coupled receptor kinase 2.
- S1P-Dependent trafficking of intracellular yersinia pestis through lymph nodes establishes Buboes and systemic infection.
- S1P₂ receptor regulation of sphingosine-1-phosphate effects on conventional outflow physiology.
- Sphingosine-1-phosphate effects on the inner wall of Schlemm's canal and outflow facility in perfused human eyes.
- Targeting blood-brain barrier sphingolipid signaling reduces basal P-glycoprotein activity and improves drug delivery to the brain.
- The DNA damage- and transcription-associated protein paxip1 controls thymocyte development and emigration.
- Up-regulating sphingosine 1-phosphate receptor-2 signaling impairs chemotactic, wound-healing, and morphogenetic responses in senescent endothelial cells.