Glucose Transport Proteins, Facilitative
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Subject Areas on Research
- Association study of genetic polymorphisms of SLC2A10 gene and type 2 diabetes in the Taiwanese population.
- Cell surface polarization during yeast mating.
- Gene expression profiling of peripheral blood leukocytes shows consistent longitudinal downregulation of TOMM40 and upregulation of KIR2DL5A, PLOD1, and SLC2A8 among fast progressors in early Alzheimer's disease.
- Genetics of hyperuricemia and gout: implications for the present and future.
- Glucose metabolism in lymphocytes is a regulated process with significant effects on immune cell function and survival.
- Intestinal, but not hepatic, ChREBP is required for fructose tolerance.
- Mitochondrial GLUT10 facilitates dehydroascorbic acid import and protects cells against oxidative stress: mechanistic insight into arterial tortuosity syndrome.
- Molecular cloning of a novel member of the GLUT family of transporters, SLC2a10 (GLUT10), localized on chromosome 20q13.1: a candidate gene for NIDDM susceptibility.
- Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
- Optimizing SGLT inhibitor treatment for diabetes with chronic kidney diseases.
- Quantitative determinants of aerobic glycolysis identify flux through the enzyme GAPDH as a limiting step.
- SLC2A10 genetic polymorphism predicts development of peripheral arterial disease in patients with type 2 diabetes. SLC2A10 and PAD in type 2 diabetes.
- The uric acid transporter SLC2A9 is a direct target gene of the tumor suppressor p53 contributing to antioxidant defense.
- Understanding intestinal glucose transporter expression in obese compared to non-obese subjects.