G Protein-Coupled Inwardly-Rectifying Potassium Channels
Subject Areas on Research
- Activation of the cloned muscarinic potassium channel by G protein beta gamma subunits.
- Evidence that direct binding of G beta gamma to the GIRK1 G protein-gated inwardly rectifying K+ channel is important for channel activation.
- Inhibitory and excitatory mechanisms underlying auditory responses to learned vocalizations in the songbird nucleus HVC.
- Molecular basis for interactions of G protein betagamma subunits with effectors.
- Pertussis-toxin-sensitive Galpha subunits selectively bind to C-terminal domain of neuronal GIRK channels: evidence for a heterotrimeric G-protein-channel complex.
- Phosphorylation and nuclear translocation of a regulator of G protein signaling (RGS10).
- Quantitative trait locus and computational mapping identifies Kcnj9 (GIRK3) as a candidate gene affecting analgesia from multiple drug classes.
- The fat mass and obesity associated gene (Fto) regulates activity of the dopaminergic midbrain circuitry.
- The plant innate immunity response in stomatal guard cells invokes G-protein-dependent ion channel regulation.