NF-kappa B p50 Subunit
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Subject Areas on Research
- Alteration of NF-kappa B p50 DNA binding kinetics by S-nitrosylation.
- Autophagy enhances NFκB activity in specific tissue macrophages by sequestering A20 to boost antifungal immunity.
- Bcl-3 acts as an innate immune modulator by controlling antimicrobial responses in keratinocytes.
- Differential effects of nitric oxide-mediated S-nitrosylation on p50 and c-jun DNA binding.
- Endotoxin-mediated S-nitrosylation of p50 alters NF-kappa B-dependent gene transcription in ANA-1 murine macrophages.
- Frequent engagement of the classical and alternative NF-kappaB pathways by diverse genetic abnormalities in multiple myeloma.
- Genome-wide association study of treatment refractory schizophrenia in Han Chinese.
- H1 RNA polymerase III promoter-driven expression of an RNA aptamer leads to high-level inhibition of intracellular protein activity.
- Inhibition of NF-kappa B by S-nitrosylation.
- Inhibition of NF-kappaB activation reduces the tissue effects of transgenic IL-13.
- Inhibition of NF-kappaB1 and NF-kappaB2 activation in prostate cancer cells treated with antibody against the carboxyl terminal domain of GRP78: effect of p53 upregulation.
- Molecular impact of selective NFKB1 and NFKB2 signaling on DLBCL phenotype.
- NF-κB p50 activation associated with immune dysregulation confers poorer survival for diffuse large B-cell lymphoma patients with wild-type p53.
- NOS2 regulation of NF-kappaB by S-nitrosylation of p65.
- Peripheral blood expression of nuclear factor-kappab-regulated genes is associated with rheumatoid arthritis disease activity and responds differentially to anti-tumor necrosis factor-alpha versus methotrexate.
- Proteasome inhibitor attenuates skeletal muscle reperfusion injury by blocking the pathway of nuclear factor-kappaB activation.
- The apolipoprotein E-mimetic peptide COG112 inhibits the inflammatory response to Citrobacter rodentium in colonic epithelial cells by preventing NF-kappaB activation.
- The necroptosis adaptor RIPK3 promotes injury-induced cytokine expression and tissue repair.
- Thioredoxin-mediated denitrosylation regulates cytokine-induced nuclear factor κB (NF-κB) activation.