gag Gene Products, Human Immunodeficiency Virus
-
Subject Areas on Research
-
A phase IIA randomized clinical trial of a multiclade HIV-1 DNA prime followed by a multiclade rAd5 HIV-1 vaccine boost in healthy adults (HVTN204).
-
A soluble inhibitor of T lymphocyte function induced by HIV-1 infection of CD4+ T cells: characterization of a cellular protein and its relationship to p15E.
-
A strongly selected mutation in the HIV-1 genome is independent of T cell responses and neutralizing antibodies.
-
Access of HIV-2 to CD169-dependent dendritic cell-mediated trans infection pathway is attenuated.
-
Basic residues in the nucleocapsid domain of Gag are critical for late events of HIV-1 budding.
-
Cellular fatty acid synthase is required for late stages of HIV-1 replication.
-
Cross-reactive potential of human T-lymphocyte responses in HIV-1 infection.
-
DNA Priming Increases Frequency of T-Cell Responses to a Vesicular Stomatitis Virus HIV Vaccine with Specific Enhancement of CD8+ T-Cell Responses by Interleukin-12 Plasmid DNA.
-
Dendritic Cell Immunotherapy for HIV-1 Infection Using Autologous HIV-1 RNA: A Randomized, Double-Blind, Placebo-Controlled Clinical Trial.
-
Developing an HIV vaccine.
-
Discovery of potential dual-target prodrugs of HIV-1 reverse transcriptase and nucleocapsid protein 7.
-
Distinct kinetics of Gag-specific CD4+ and CD8+ T cell responses during acute HIV-1 infection.
-
Drug-associated changes in amino acid residues in Gag p2, p7(NC), and p6(Gag)/p6(Pol) in human immunodeficiency virus type 1 (HIV-1) display a dominant effect on replicative fitness and drug response.
-
Evaluation of humoral, mucosal, and cellular immune responses following co-immunization of HIV-1 Gag and Env proteins expressed by Newcastle disease virus.
-
GB virus type C envelope protein E2 elicits antibodies that react with a cellular antigen on HIV-1 particles and neutralize diverse HIV-1 isolates.
-
Gene deletions in Mycobacterium bovis BCG stimulate increased CD8+ T cell responses.
-
Genetic determinants in HIV-1 Gag and Env V3 are related to viral response to combination antiretroviral therapy with a protease inhibitor.
-
Genetic variability of the U5 and downstream sequence of major HIV-1 subtypes and circulating recombinant forms.
-
Impact of immune escape mutations on HIV-1 fitness in the context of the cognate transmitted/founder genome.
-
Inhibition of protein kinase C by a peptide conjugate homologous to a domain of the retroviral protein p15E.
-
Intradermal HIV-1 DNA Immunization Using Needle-Free Zetajet Injection Followed by HIV-Modified Vaccinia Virus Ankara Vaccination Is Safe and Immunogenic in Mozambican Young Adults: A Phase I Randomized Controlled Trial.
-
Mosaic vaccines elicit CD8+ T lymphocyte responses that confer enhanced immune coverage of diverse HIV strains in monkeys.
-
Mucosal Immunization with Newcastle Disease Virus Vector Coexpressing HIV-1 Env and Gag Proteins Elicits Potent Serum, Mucosal, and Cellular Immune Responses That Protect against Vaccinia Virus Env and Gag Challenges.
-
Natural variation in HIV-1 protease, Gag p7 and p6, and protease cleavage sites within gag/pol polyproteins: amino acid substitutions in the absence of protease inhibitors in mothers and children infected by human immunodeficiency virus type 1.
-
Naturally occurring amino acid polymorphisms in human immunodeficiency virus type 1 (HIV-1) Gag p7(NC) and the C-cleavage site impact Gag-Pol processing by HIV-1 protease.
-
New betulinic acid derivatives for bevirimat-resistant human immunodeficiency virus type-1.
-
Potential To Streamline Heterologous DNA Prime and NYVAC/Protein Boost HIV Vaccine Regimens in Rhesus Macaques by Employing Improved Antigens.
-
Preexisting compensatory amino acids compromise fitness costs of a HIV-1 T cell escape mutation.
-
Rapid memory CD8+ T-lymphocyte induction through priming with recombinant Mycobacterium smegmatis.
-
The nucleocapsid region of HIV-1 Gag cooperates with the PTAP and LYPXnL late domains to recruit the cellular machinery necessary for viral budding.
-
Virus particle release from glycosphingolipid-enriched microdomains is essential for dendritic cell-mediated capture and transfer of HIV-1 and henipavirus.
-
Keywords of People