Extracellular Traps

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  Subject Areas on Research

  • Altered Mitochondrial Homeostasis during Systemic Lupus Erythematosus Impairs Neutrophil Extracellular Trap Formation Rendering Neutrophils Ineffective at Combating Staphylococcus aureus. 
  • Gout, tophi and the wonders of NETs. 
  • IL-1β, IL-18, and eicosanoids promote neutrophil recruitment to pore-induced intracellular traps following pyroptosis. 
  • IL26, a Noncanonical Mediator of DNA Inflammatory Stimulation, Promotes TNBC Engraftment and Progression in Association with Neutrophils. 
  • Moonlighting chromatin: when DNA escapes nuclear control. 
  • Myeloid-Specific Deletion of Peptidylarginine Deiminase 4 Mitigates Atherosclerosis. 
  • Neutrophil accumulation and NET release contribute to thrombosis in HIT. 
  • Neutrophil extracellular traps are pathogenic in primary graft dysfunction after lung transplantation. 
  • Neutrophil functional heterogeneity is a fixed phenotype and is associated with distinct gene expression profiles. 
  • Neutrophil-Related Gene Expression and Low-Density Granulocytes Associated With Disease Activity and Response to Treatment in Antineutrophil Cytoplasmic Antibody-Associated Vasculitis. 
  • Neutrophils Create an ImpeNETrable Shield between Tumor and Cytotoxic Immune Cells. 
  • Sepsis-Induced Coagulopathy and Disseminated Intravascular Coagulation. 
  • The Cutaneous Wound Innate Immunological Microenvironment. 
  • The influence of hyperglycemia on neutrophil extracellular trap formation and endothelial glycocalyx damage in a mouse model of type 2 diabetes. 
  • Transient Bacteremia Promotes Catheter-Related Central Venous Thrombosis through Neutrophil Extracellular Traps.