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An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1.

Publication ,  Journal Article
Bonsignori, M; Wiehe, K; Grimm, SK; Lynch, R; Yang, G; Kozink, DM; Perrin, F; Cooper, AJ; Hwang, K-K; Chen, X; Liu, M; McKee, K; Parks, RJ ...
Published in: J Clin Invest
April 2014

Broadly HIV-1-neutralizing antibodies (BnAbs) display one or more unusual traits, including a long heavy chain complementarity-determining region 3 (HCDR3), polyreactivity, and high levels of somatic mutations. These shared characteristics suggest that BnAb development might be limited by immune tolerance controls. It has been postulated that HIV-1-infected individuals with autoimmune disease and defective immune tolerance mechanisms may produce BnAbs more readily than those without autoimmune diseases. In this study, we identified an HIV-1-infected individual with SLE who exhibited controlled viral load (<5,000 copies/ml) in the absence of controlling HLA phenotypes and developed plasma HIV-1 neutralization breadth. We collected memory B cells from this individual and isolated a BnAb, CH98, that targets the CD4 binding site (CD4bs) of HIV-1 envelope glycoprotein 120 (gp120). CH98 bound to human antigens including dsDNA, which is specifically associated with SLE. Anti-dsDNA reactivity was also present in the patient's plasma. CH98 had a mutation frequency of 25% and 15% nt somatic mutations in the heavy and light chain variable domains, respectively, a long HCDR3, and a deletion in the light chain CDR1. The occurrence of anti-dsDNA reactivity by a HIV-1 CD4bs BnAb in an individual with SLE raises the possibility that some BnAbs and SLE-associated autoantibodies arise from similar pools of B cells.

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Published In

J Clin Invest

DOI

EISSN

1558-8238

Publication Date

April 2014

Volume

124

Issue

4

Start / End Page

1835 / 1843

Location

United States

Related Subject Headings

  • Viral Load
  • Sequence Homology, Nucleic Acid
  • Sequence Homology, Amino Acid
  • Protein Conformation
  • Mutation
  • Multiprotein Complexes
  • Molecular Sequence Data
  • Models, Molecular
  • Lupus Erythematosus, Systemic
  • Immunology
 

Citation

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MLA
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Bonsignori, M., Wiehe, K., Grimm, S. K., Lynch, R., Yang, G., Kozink, D. M., … Haynes, B. F. (2014). An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1. J Clin Invest, 124(4), 1835–1843. https://doi.org/10.1172/JCI73441
Bonsignori, Mattia, Kevin Wiehe, Sebastian K. Grimm, Rebecca Lynch, Guang Yang, Daniel M. Kozink, Florence Perrin, et al. “An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1.J Clin Invest 124, no. 4 (April 2014): 1835–43. https://doi.org/10.1172/JCI73441.
Bonsignori M, Wiehe K, Grimm SK, Lynch R, Yang G, Kozink DM, et al. An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1. J Clin Invest. 2014 Apr;124(4):1835–43.
Bonsignori, Mattia, et al. “An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1.J Clin Invest, vol. 124, no. 4, Apr. 2014, pp. 1835–43. Pubmed, doi:10.1172/JCI73441.
Bonsignori M, Wiehe K, Grimm SK, Lynch R, Yang G, Kozink DM, Perrin F, Cooper AJ, Hwang K-K, Chen X, Liu M, McKee K, Parks RJ, Eudailey J, Wang M, Clowse M, Criscione-Schreiber LG, Moody MA, Ackerman ME, Boyd SD, Gao F, Kelsoe G, Verkoczy L, Tomaras GD, Liao H-X, Kepler TB, Montefiori DC, Mascola JR, Haynes BF. An autoreactive antibody from an SLE/HIV-1 individual broadly neutralizes HIV-1. J Clin Invest. 2014 Apr;124(4):1835–1843.

Published In

J Clin Invest

DOI

EISSN

1558-8238

Publication Date

April 2014

Volume

124

Issue

4

Start / End Page

1835 / 1843

Location

United States

Related Subject Headings

  • Viral Load
  • Sequence Homology, Nucleic Acid
  • Sequence Homology, Amino Acid
  • Protein Conformation
  • Mutation
  • Multiprotein Complexes
  • Molecular Sequence Data
  • Models, Molecular
  • Lupus Erythematosus, Systemic
  • Immunology