Antibody light-chain-restricted recognition of the site of immune pressure in the RV144 HIV-1 vaccine trial is phylogenetically conserved.
Journal Article (Journal Article)
In HIV-1, the ability to mount antibody responses to conserved, neutralizing epitopes is critical for protection. Here we have studied the light chain usage of human and rhesus macaque antibodies targeted to a dominant region of the HIV-1 envelope second variable (V2) region involving lysine (K) 169, the site of immune pressure in the RV144 vaccine efficacy trial. We found that humans and rhesus macaques used orthologous lambda variable gene segments encoding a glutamic acid-aspartic acid (ED) motif for K169 recognition. Structure determination of an unmutated ancestor antibody demonstrated that the V2 binding site was preconfigured for ED motif-mediated recognition prior to maturation. Thus, light chain usage for recognition of the site of immune pressure in the RV144 trial is highly conserved across species. These data indicate that the HIV-1 K169-recognizing ED motif has persisted over the diversification between rhesus macaques and humans, suggesting an evolutionary advantage of this antibody recognition mode.
Full Text
Duke Authors
- Alam, S. Munir
- Haynes, Barton Ford
- Kelsoe, Garnett H.
- Montefiori, David Charles
- Moody, Michael Anthony
- Sekaran, Moses
- Tomaras, Georgia Doris
- Wiehe, Kevin J
Cited Authors
- Wiehe, K; Easterhoff, D; Luo, K; Nicely, NI; Bradley, T; Jaeger, FH; Dennison, SM; Zhang, R; Lloyd, KE; Stolarchuk, C; Parks, R; Sutherland, LL; Scearce, RM; Morris, L; Kaewkungwal, J; Nitayaphan, S; Pitisuttithum, P; Rerks-Ngarm, S; Sinangil, F; Phogat, S; Michael, NL; Kim, JH; Kelsoe, G; Montefiori, DC; Tomaras, GD; Bonsignori, M; Santra, S; Kepler, TB; Alam, SM; Moody, MA; Liao, H-X; Haynes, BF
Published Date
- December 18, 2014
Published In
Volume / Issue
- 41 / 6
Start / End Page
- 909 - 918
PubMed ID
- 25526306
Pubmed Central ID
- PMC4324565
Electronic International Standard Serial Number (EISSN)
- 1097-4180
Digital Object Identifier (DOI)
- 10.1016/j.immuni.2014.11.014
Language
- eng
Conference Location
- United States