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PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1.

Publication ,  Journal Article
Siska, PJ; Rathmell, JC
Published in: Molecular cell
June 2015

In this issue, Lee et al. (2015) show that PKC directly phosphorylates the glucose transporter Glut1, in order to promote glucose uptake in response to growth factor signaling.

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Published In

Molecular cell

DOI

EISSN

1097-4164

ISSN

1097-2765

Publication Date

June 2015

Volume

58

Issue

5

Start / End Page

711 / 712

Related Subject Headings

  • Protein Kinase C-alpha
  • Monosaccharide Transport Proteins
  • Humans
  • Glucose Transporter Type 1
  • Developmental Biology
  • Carbohydrate Metabolism, Inborn Errors
  • Animals
  • 42 Health sciences
  • 32 Biomedical and clinical sciences
  • 31 Biological sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Siska, P. J., & Rathmell, J. C. (2015). PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1. Molecular Cell, 58(5), 711–712. https://doi.org/10.1016/j.molcel.2015.05.025
Siska, Peter J., and Jeffrey C. Rathmell. “PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1.Molecular Cell 58, no. 5 (June 2015): 711–12. https://doi.org/10.1016/j.molcel.2015.05.025.
Siska PJ, Rathmell JC. PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1. Molecular cell. 2015 Jun;58(5):711–2.
Siska, Peter J., and Jeffrey C. Rathmell. “PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1.Molecular Cell, vol. 58, no. 5, June 2015, pp. 711–12. Epmc, doi:10.1016/j.molcel.2015.05.025.
Siska PJ, Rathmell JC. PKCs Sweeten Cell Metabolism by Phosphorylation of Glut1. Molecular cell. 2015 Jun;58(5):711–712.
Journal cover image

Published In

Molecular cell

DOI

EISSN

1097-4164

ISSN

1097-2765

Publication Date

June 2015

Volume

58

Issue

5

Start / End Page

711 / 712

Related Subject Headings

  • Protein Kinase C-alpha
  • Monosaccharide Transport Proteins
  • Humans
  • Glucose Transporter Type 1
  • Developmental Biology
  • Carbohydrate Metabolism, Inborn Errors
  • Animals
  • 42 Health sciences
  • 32 Biomedical and clinical sciences
  • 31 Biological sciences