On the driving force for fatique crack formation from inclusion and voids in a cast A356 aluminum alloy

Published

Journal Article

Monotonic and cyclic finite element simulations are conducted on linear-elastic inclusions and voids embedded in an elasto-plastic matrix material. The elasto-plastic material is modeled with both kinematic and isotropic hardening laws cast in a hardening minus recovery format. Three loading amplitudes (Δε/2 = 0.10%, 0.15, 0.20%) and three load ratios (R=-1, 0, 0.5) are considered. From a continuum standpoint, the primary driving force for fatigue crack formation is assumed to be the local maximum plastic shear strain range. Δγmax, with respect to all possible shear strain planes. For certain inhomogeneities, the Δγmax was as high as ten times the far field strains. Bonded inclusions have Δγmax values two orders of magnitude smaller than voids, cracked, or debonded inclusions. A cracked inclusion facilitates extremely large local stresses in the broken particle halves, which will invariably facilitate the debonding of a cracked particle. Based on these two observations, debonded inclusions and voids are asserted to be the critical inhomogeneities for fatigue crack formation. Furthermore, for voids and debonded inclusions, shape has a negligible effect on fatigue crack formation compared to other significant effects such as inhomogeneity size and reserved loading conditions (R ratio). Increasing the size of an inclusion by a factor of four increases Δγmax by about a factor of two. At low R ratios (-1) equivalent sized voids and debonded inclusions have comparable Δγmax values. At higher R ratios (0, 0.5) debonded inclusions have Δγmax values twice that of voids.

Full Text

Duke Authors

Cited Authors

  • Gall, K; Horstemeyer, MF; Degner, BW; McDowell, DL; Fan, J

Published Date

  • January 1, 2001

Published In

Volume / Issue

  • 108 / 3

Start / End Page

  • 207 - 233

International Standard Serial Number (ISSN)

  • 0376-9429

Digital Object Identifier (DOI)

  • 10.1023/A:1011033304600

Citation Source

  • Scopus