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Gln attenuates inflammation following sepsis through loss of Cul-1 neddylation and thereby inhibits NF-kappa B activation and IKB-alpha degredation

Publication ,  Conference
Singleton, K; Wischmeyer, P
Published in: SHOCK
June 1, 2007
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Duke Scholars

Paul Edmund Wischmeyer
Author Paul Edmund Wischmeyer Anesthesiology, Critical Care Medicine

Published In

SHOCK

ISSN

1073-2322

Publication Date

June 1, 2007

Volume

27

Start / End Page

41 / 41

Location

Baltimore, MD

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Conference Name

30th Annual Conference of the Shock-Society

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences
 

Citation

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Singleton, K., & Wischmeyer, P. (2007). Gln attenuates inflammation following sepsis through loss of Cul-1 neddylation and thereby inhibits NF-kappa B activation and IKB-alpha degredation. In SHOCK (Vol. 27, pp. 41–41). Baltimore, MD: LIPPINCOTT WILLIAMS & WILKINS.
Singleton, K., and P. Wischmeyer. “Gln attenuates inflammation following sepsis through loss of Cul-1 neddylation and thereby inhibits NF-kappa B activation and IKB-alpha degredation.” In SHOCK, 27:41–41. LIPPINCOTT WILLIAMS & WILKINS, 2007.
Singleton, K., and P. Wischmeyer. “Gln attenuates inflammation following sepsis through loss of Cul-1 neddylation and thereby inhibits NF-kappa B activation and IKB-alpha degredation.” SHOCK, vol. 27, LIPPINCOTT WILLIAMS & WILKINS, 2007, pp. 41–41.

Published In

SHOCK

ISSN

1073-2322

Publication Date

June 1, 2007

Volume

27

Start / End Page

41 / 41

Location

Baltimore, MD

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Conference Name

30th Annual Conference of the Shock-Society

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences