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Glutamine treatment increases cell viabilily in heat stressed murine embryonic fibroblast cells via activation of the O-linked-N-acetyglucosamine pathway

Publication ,  Conference
Hamiel, C; Pinto, S; Singleton, K; Wischmeyer, PE
Published in: SHOCK
June 1, 2007

Duke Scholars

Published In

SHOCK

ISSN

1073-2322

Publication Date

June 1, 2007

Volume

27

Start / End Page

36 / 37

Location

Baltimore, MD

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Conference Name

30th Annual Conference of the Shock-Society

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences
 

Citation

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MLA
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Hamiel, C., Pinto, S., Singleton, K., & Wischmeyer, P. E. (2007). Glutamine treatment increases cell viabilily in heat stressed murine embryonic fibroblast cells via activation of the O-linked-N-acetyglucosamine pathway. In SHOCK (Vol. 27, pp. 36–37). Baltimore, MD: LIPPINCOTT WILLIAMS & WILKINS.
Hamiel, C., S. Pinto, K. Singleton, and P. E. Wischmeyer. “Glutamine treatment increases cell viabilily in heat stressed murine embryonic fibroblast cells via activation of the O-linked-N-acetyglucosamine pathway.” In SHOCK, 27:36–37. LIPPINCOTT WILLIAMS & WILKINS, 2007.
Hamiel C, Pinto S, Singleton K, Wischmeyer PE. Glutamine treatment increases cell viabilily in heat stressed murine embryonic fibroblast cells via activation of the O-linked-N-acetyglucosamine pathway. In: SHOCK. LIPPINCOTT WILLIAMS & WILKINS; 2007. p. 36–7.
Hamiel C, Pinto S, Singleton K, Wischmeyer PE. Glutamine treatment increases cell viabilily in heat stressed murine embryonic fibroblast cells via activation of the O-linked-N-acetyglucosamine pathway. SHOCK. LIPPINCOTT WILLIAMS & WILKINS; 2007. p. 36–37.

Published In

SHOCK

ISSN

1073-2322

Publication Date

June 1, 2007

Volume

27

Start / End Page

36 / 37

Location

Baltimore, MD

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Conference Name

30th Annual Conference of the Shock-Society

Related Subject Headings

  • Emergency & Critical Care Medicine
  • 3202 Clinical sciences
  • 1103 Clinical Sciences