Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD
Publication
, Conference
Volkow, N; Wang, G-J; Tomasi, D; Kollins, S; Wigal, T; Newcorn, J; Telang, F; Fowler, J; Logan, J; Swanson, J
Published in: JOURNAL OF NUCLEAR MEDICINE
2011
Duke Scholars
Published In
JOURNAL OF NUCLEAR MEDICINE
EISSN
1535-5667
ISSN
0161-5505
Publication Date
2011
Volume
52
Related Subject Headings
- Nuclear Medicine & Medical Imaging
- 3202 Clinical sciences
- 1103 Clinical Sciences
Citation
APA
Chicago
ICMJE
MLA
NLM
Volkow, N., Wang, G.-J., Tomasi, D., Kollins, S., Wigal, T., Newcorn, J., … Swanson, J. (2011). Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD. In JOURNAL OF NUCLEAR MEDICINE (Vol. 52).
Volkow, Nora, Gene-Jack Wang, Dardo Tomasi, Scott Kollins, Tim Wigal, Jeffrey Newcorn, Frank Telang, Joanna Fowler, Jean Logan, and James Swanson. “Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD.” In JOURNAL OF NUCLEAR MEDICINE, Vol. 52, 2011.
Volkow N, Wang G-J, Tomasi D, Kollins S, Wigal T, Newcorn J, et al. Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD. In: JOURNAL OF NUCLEAR MEDICINE. 2011.
Volkow, Nora, et al. “Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD.” JOURNAL OF NUCLEAR MEDICINE, vol. 52, 2011.
Volkow N, Wang G-J, Tomasi D, Kollins S, Wigal T, Newcorn J, Telang F, Fowler J, Logan J, Swanson J. Methylphenidate induced dopamine increases in striatum and temporal and frontal cortices predicts response to treatment in ADHD. JOURNAL OF NUCLEAR MEDICINE. 2011.
Published In
JOURNAL OF NUCLEAR MEDICINE
EISSN
1535-5667
ISSN
0161-5505
Publication Date
2011
Volume
52
Related Subject Headings
- Nuclear Medicine & Medical Imaging
- 3202 Clinical sciences
- 1103 Clinical Sciences