Corrosion Resistance of 3D Printed Ti6Al4V Gyroid Lattices with Varying Porosity.

Journal Article (Journal Article)

Corrosion of medical implants is a possible failure mode via induced local inflammatory effects, systemic deposition and corrosion related mechanical failure. Cyclic potentiodynamic polarisation (CPP) testing was utilized to evaluate the effect of increased porosity (60% and 80%) and decreased wall thickness in gyroid lattice structures on the electrochemical behaviour of LPBF Ti6Al4V structures. The use of CPP allowed for the landmarks of breakdown potential, resting potential and vertex potential to be analysed, as well as facilitating the construction of Tafel plots and qualitative Goldberg analysis. The results indicated that 60% gyroid samples were most susceptible to the onset of pitting corrosion when compared to 80% gyroid and solid samples. This was shown through decreased breakdown and vertex potentials and were found to correlate to increased lattice surface area to void volume ratio. Tafel plots indicated that despite the earlier onset of pitting corrosion, both gyroid test groups displayed lower rates of corrosion per year, indicating a lower severity of corrosion. This study highlighted inherent tradeoffs between lattice optimisation and corrosion behaviour with a potential parabolic link between void volume, surface area and corrosion being identified. This potential link is supported by 60% gyroid samples having the lowest breakdown potentials, but investigation into other porosity ranges is suggested to support the hypothesis. All 3D printed materials studied here showed breakdown potentials higher than ASTM F2129's suggestion of 800 mV for evaluation within the physiological environment, indicating that under static conditions pitting and crevice corrosion should not initiate within the body.

Full Text

Duke Authors

Cited Authors

  • Sharp, R; Pelletier, MH; Walsh, WR; Kelly, CN; Gall, K

Published Date

  • July 2022

Published In

Volume / Issue

  • 15 / 14

Start / End Page

  • 4805 -

PubMed ID

  • 35888273

Pubmed Central ID

  • PMC9316743

Electronic International Standard Serial Number (EISSN)

  • 1996-1944

International Standard Serial Number (ISSN)

  • 1996-1944

Digital Object Identifier (DOI)

  • 10.3390/ma15144805


  • eng