Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma.
Publication
, Journal Article
Ingram, JL; Rice, A; Geisenhoffer, K; Madtes, DK; Bonner, JC
Published in: Chest
March 2003
Duke Scholars
Published In
Chest
DOI
ISSN
0012-3692
Publication Date
March 2003
Volume
123
Issue
3 Suppl
Start / End Page
422S / 424S
Location
United States
Related Subject Headings
- Transcriptional Activation
- Signal Transduction
- Respiratory System
- Rats, Sprague-Dawley
- Rats
- Pulmonary Fibrosis
- Myofibrils
- Lung
- Interleukin-13
- In Vitro Techniques
Citation
APA
Chicago
ICMJE
MLA
NLM
Ingram, J. L., Rice, A., Geisenhoffer, K., Madtes, D. K., & Bonner, J. C. (2003). Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma. Chest, 123(3 Suppl), 422S-424S. https://doi.org/10.1378/chest.123.3_suppl.422s
Ingram, Jennifer L., Annette Rice, Kristen Geisenhoffer, David K. Madtes, and James C. Bonner. “Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma.” Chest 123, no. 3 Suppl (March 2003): 422S-424S. https://doi.org/10.1378/chest.123.3_suppl.422s.
Ingram JL, Rice A, Geisenhoffer K, Madtes DK, Bonner JC. Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma. Chest. 2003 Mar;123(3 Suppl):422S-424S.
Ingram, Jennifer L., et al. “Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma.” Chest, vol. 123, no. 3 Suppl, Mar. 2003, pp. 422S-424S. Pubmed, doi:10.1378/chest.123.3_suppl.422s.
Ingram JL, Rice A, Geisenhoffer K, Madtes DK, Bonner JC. Interleukin-13 stimulates the proliferation of lung myofibroblasts via a signal transducer and activator of transcription-6-dependent mechanism: a possible mechanism for the development of airway fibrosis in asthma. Chest. 2003 Mar;123(3 Suppl):422S-424S.
Published In
Chest
DOI
ISSN
0012-3692
Publication Date
March 2003
Volume
123
Issue
3 Suppl
Start / End Page
422S / 424S
Location
United States
Related Subject Headings
- Transcriptional Activation
- Signal Transduction
- Respiratory System
- Rats, Sprague-Dawley
- Rats
- Pulmonary Fibrosis
- Myofibrils
- Lung
- Interleukin-13
- In Vitro Techniques