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Beta-arrestins: multifunctional cellular mediators.

Publication ,  Journal Article
Barki-Harrington, L; Rockman, HA
Published in: Physiology (Bethesda)
February 2008

Initially thought to play a role only in G-protein-coupled receptor desensitization, beta-arrestins are ascribed with new roles such as scaffolding and signaling proteins by their own right. This review explores the many functions of beta-arrestins, with an emphasis on their recently identified role as regulators of receptor signaling.

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Published In

Physiology (Bethesda)

DOI

ISSN

1548-9213

Publication Date

February 2008

Volume

23

Start / End Page

17 / 22

Location

United States

Related Subject Headings

  • beta-Arrestins
  • Signal Transduction
  • Receptors, G-Protein-Coupled
  • Protein Transport
  • Protein Processing, Post-Translational
  • Physiology
  • Myocardium
  • Ligands
  • Humans
  • G-Protein-Coupled Receptor Kinases
 

Citation

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Barki-Harrington, L., & Rockman, H. A. (2008). Beta-arrestins: multifunctional cellular mediators. Physiology (Bethesda), 23, 17–22. https://doi.org/10.1152/physiol.00042.2007
Barki-Harrington, Liza, and Howard A. Rockman. “Beta-arrestins: multifunctional cellular mediators.Physiology (Bethesda) 23 (February 2008): 17–22. https://doi.org/10.1152/physiol.00042.2007.
Barki-Harrington L, Rockman HA. Beta-arrestins: multifunctional cellular mediators. Physiology (Bethesda). 2008 Feb;23:17–22.
Barki-Harrington, Liza, and Howard A. Rockman. “Beta-arrestins: multifunctional cellular mediators.Physiology (Bethesda), vol. 23, Feb. 2008, pp. 17–22. Pubmed, doi:10.1152/physiol.00042.2007.
Barki-Harrington L, Rockman HA. Beta-arrestins: multifunctional cellular mediators. Physiology (Bethesda). 2008 Feb;23:17–22.

Published In

Physiology (Bethesda)

DOI

ISSN

1548-9213

Publication Date

February 2008

Volume

23

Start / End Page

17 / 22

Location

United States

Related Subject Headings

  • beta-Arrestins
  • Signal Transduction
  • Receptors, G-Protein-Coupled
  • Protein Transport
  • Protein Processing, Post-Translational
  • Physiology
  • Myocardium
  • Ligands
  • Humans
  • G-Protein-Coupled Receptor Kinases