Capsaicin
Publication
, Journal Article
Ochs, G; Liedtke, W
Published in: Aktuelle Neurologie
January 1, 1995
Capsaicin binds and acts at the vanilloid receptor, and causes initial excitatory actions. After long-term application, desensitisation of pain afferents is observed. Therapeutic trials and clinical studies have been performed with different forms of neuralgias. The analgetic effect has been substantiated for post-herpetic neuralgia and painful diabetic neuralgia. Other indications, such as neurogenic bladder disturbance, cluster headache, and pruritus are still in the experimental stage.
Duke Scholars
Published In
Aktuelle Neurologie
DOI
ISSN
0302-4350
Publication Date
January 1, 1995
Volume
22
Issue
4
Start / End Page
164 / 166
Related Subject Headings
- Neurology & Neurosurgery
- 4201 Allied health and rehabilitation science
- 3209 Neurosciences
- 3202 Clinical sciences
- 1109 Neurosciences
- 1103 Clinical Sciences
Citation
APA
Chicago
ICMJE
MLA
NLM
Ochs, G., & Liedtke, W. (1995). Capsaicin. Aktuelle Neurologie, 22(4), 164–166. https://doi.org/10.1055/s-2007-1017914
Ochs, G., and W. Liedtke. “Capsaicin.” Aktuelle Neurologie 22, no. 4 (January 1, 1995): 164–66. https://doi.org/10.1055/s-2007-1017914.
Ochs G, Liedtke W. Capsaicin. Aktuelle Neurologie. 1995 Jan 1;22(4):164–6.
Ochs, G., and W. Liedtke. “Capsaicin.” Aktuelle Neurologie, vol. 22, no. 4, Jan. 1995, pp. 164–66. Scopus, doi:10.1055/s-2007-1017914.
Ochs G, Liedtke W. Capsaicin. Aktuelle Neurologie. 1995 Jan 1;22(4):164–166.
Published In
Aktuelle Neurologie
DOI
ISSN
0302-4350
Publication Date
January 1, 1995
Volume
22
Issue
4
Start / End Page
164 / 166
Related Subject Headings
- Neurology & Neurosurgery
- 4201 Allied health and rehabilitation science
- 3209 Neurosciences
- 3202 Clinical sciences
- 1109 Neurosciences
- 1103 Clinical Sciences