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Calcium channel modulation as an all-purpose mechanism for short-term synaptic plasticity.

Publication ,  Journal Article
Cheng, Q; Augustine, GJ
Published in: Neuron
January 2008

The paper by Mochida et al. in this issue of Neuron proposes that synaptic facilitation, depression, and augmentation all arise from calcium-dependent regulation of calcium channels in the presynaptic terminal. Their proposal provides a unifying explanation for several forms of short-term presynaptic plasticity.

Published In

Neuron

DOI

EISSN

1097-4199

ISSN

0896-6273

Publication Date

January 2008

Volume

57

Issue

2

Start / End Page

171 / 172

Related Subject Headings

  • Synapses
  • Neuronal Plasticity
  • Neurology & Neurosurgery
  • Calcium Channels
  • Animals
  • 5202 Biological psychology
  • 3209 Neurosciences
  • 1702 Cognitive Sciences
  • 1701 Psychology
  • 1109 Neurosciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Cheng, Q., & Augustine, G. J. (2008). Calcium channel modulation as an all-purpose mechanism for short-term synaptic plasticity. Neuron, 57(2), 171–172. https://doi.org/10.1016/j.neuron.2008.01.004
Cheng, Qing, and George J. Augustine. “Calcium channel modulation as an all-purpose mechanism for short-term synaptic plasticity.Neuron 57, no. 2 (January 2008): 171–72. https://doi.org/10.1016/j.neuron.2008.01.004.
Cheng, Qing, and George J. Augustine. “Calcium channel modulation as an all-purpose mechanism for short-term synaptic plasticity.Neuron, vol. 57, no. 2, Jan. 2008, pp. 171–72. Epmc, doi:10.1016/j.neuron.2008.01.004.
Journal cover image

Published In

Neuron

DOI

EISSN

1097-4199

ISSN

0896-6273

Publication Date

January 2008

Volume

57

Issue

2

Start / End Page

171 / 172

Related Subject Headings

  • Synapses
  • Neuronal Plasticity
  • Neurology & Neurosurgery
  • Calcium Channels
  • Animals
  • 5202 Biological psychology
  • 3209 Neurosciences
  • 1702 Cognitive Sciences
  • 1701 Psychology
  • 1109 Neurosciences