Scholars@Duke publication: 748: Mice lacking G protein-coupled receptor kinase 6 (GRK6) exhibit an enhanced uterine contractile phenotype resulting in stillbirth

748: Mice lacking G protein-coupled receptor kinase 6 (GRK6) exhibit an enhanced uterine contractile phenotype resulting in stillbirth

Publication , Conference

Grotegut, C; Mao, L; Heine, RP; Murtha, A; Rockman, H

Published in: American Journal of Obstetrics and Gynecology

January 2014

American Journal of Obstetrics and Gynecology

0002-9378

January 2014

210

S367 / S368

Elsevier BV

APA

Chicago

ICMJE

MLA

NLM

Grotegut, C., Mao, L., Heine, R. P., Murtha, A., & Rockman, H. (2014). 748: Mice lacking G protein-coupled receptor kinase 6 (GRK6) exhibit an enhanced uterine contractile phenotype resulting in stillbirth. In American Journal of Obstetrics and Gynecology (Vol. 210, pp. S367–S368). Elsevier BV. https://doi.org/10.1016/j.ajog.2013.10.781

Grotegut, Chad, Lan Mao, R Phillips Heine, Amy Murtha, and Howard Rockman. “748: Mice lacking G protein-coupled receptor kinase 6 (GRK6) exhibit an enhanced uterine contractile phenotype resulting in stillbirth.” In American Journal of Obstetrics and Gynecology, 210:S367–68. Elsevier BV, 2014. https://doi.org/10.1016/j.ajog.2013.10.781.

Grotegut, Chad, et al. “748: Mice lacking G protein-coupled receptor kinase 6 (GRK6) exhibit an enhanced uterine contractile phenotype resulting in stillbirth.” American Journal of Obstetrics and Gynecology, vol. 210, no. 1, Elsevier BV, 2014, pp. S367–68. Crossref, doi:10.1016/j.ajog.2013.10.781.