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The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice

Publication ,  Conference
Coch, R; Campbell, J; D'Alessio, DA
Published in: DIABETES
June 1, 2017

Duke Scholars

Published In

DIABETES

EISSN

1939-327X

ISSN

0012-1797

Publication Date

June 1, 2017

Volume

66

Start / End Page

A349 / A349

Location

San Diego, CA

Publisher

AMER DIABETES ASSOC

Conference Name

77th Scientific Sessions of the American-Diabetes-Association

Related Subject Headings

  • Endocrinology & Metabolism
  • 32 Biomedical and clinical sciences
  • 11 Medical and Health Sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Coch, R., Campbell, J., & D’Alessio, D. A. (2017). The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice. In DIABETES (Vol. 66, pp. A349–A349). San Diego, CA: AMER DIABETES ASSOC.
Coch, Reilly, Jonathan Campbell, and David A. D’Alessio. “The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice.” In DIABETES, 66:A349–A349. AMER DIABETES ASSOC, 2017.
Coch R, Campbell J, D’Alessio DA. The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice. In: DIABETES. AMER DIABETES ASSOC; 2017. p. A349–A349.
Coch, Reilly, et al. “The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice.” DIABETES, vol. 66, AMER DIABETES ASSOC, 2017, pp. A349–A349.
Coch R, Campbell J, D’Alessio DA. The Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitor Dapagliflozin Causes Ketosis In Nondiabetic and Diabetic Mice. DIABETES. AMER DIABETES ASSOC; 2017. p. A349–A349.

Published In

DIABETES

EISSN

1939-327X

ISSN

0012-1797

Publication Date

June 1, 2017

Volume

66

Start / End Page

A349 / A349

Location

San Diego, CA

Publisher

AMER DIABETES ASSOC

Conference Name

77th Scientific Sessions of the American-Diabetes-Association

Related Subject Headings

  • Endocrinology & Metabolism
  • 32 Biomedical and clinical sciences
  • 11 Medical and Health Sciences