A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers.
Publication
, Journal Article
Chen, J; Kastan, MB
Published in: Oncoscience
September 2017
Duke Scholars
Published In
Oncoscience
DOI
ISSN
2331-4737
Publication Date
September 2017
Volume
4
Issue
9-10
Start / End Page
122 / 123
Location
United States
Citation
APA
Chicago
ICMJE
MLA
NLM
Chen, J., & Kastan, M. B. (2017). A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers. Oncoscience, 4(9–10), 122–123. https://doi.org/10.18632/oncoscience.367
Chen, Jing, and Michael B. Kastan. “A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers.” Oncoscience 4, no. 9–10 (September 2017): 122–23. https://doi.org/10.18632/oncoscience.367.
Chen J, Kastan MB. A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers. Oncoscience. 2017 Sep;4(9–10):122–3.
Chen, Jing, and Michael B. Kastan. “A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers.” Oncoscience, vol. 4, no. 9–10, Sept. 2017, pp. 122–23. Pubmed, doi:10.18632/oncoscience.367.
Chen J, Kastan MB. A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers. Oncoscience. 2017 Sep;4(9–10):122–123.
Published In
Oncoscience
DOI
ISSN
2331-4737
Publication Date
September 2017
Volume
4
Issue
9-10
Start / End Page
122 / 123
Location
United States