Skip to main content
Journal cover image

An IRKO in the Podo: impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy.

Publication ,  Journal Article
Gurley, SB; Coffman, TM
Published in: Cell Metab
October 6, 2010

Despite its high prevalence, the mechanisms causing diabetic kidney disease remain poorly understood. In this issue of Cell Metabolism, Welsh et al. (2010) show that elimination of insulin receptors from the glomerular podocyte, a cell that is central to the pathogenesis of proteinuric renal diseases, recapitulates many features of human diabetic nephropathy.

Duke Scholars

Published In

Cell Metab

DOI

EISSN

1932-7420

Publication Date

October 6, 2010

Volume

12

Issue

4

Start / End Page

311 / 312

Location

United States

Related Subject Headings

  • Endocrinology & Metabolism
  • 3205 Medical biochemistry and metabolomics
  • 3101 Biochemistry and cell biology
  • 1101 Medical Biochemistry and Metabolomics
  • 0601 Biochemistry and Cell Biology
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Gurley, S. B., & Coffman, T. M. (2010). An IRKO in the Podo: impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy. Cell Metab, 12(4), 311–312. https://doi.org/10.1016/j.cmet.2010.09.007
Gurley, Susan B., and Thomas M. Coffman. “An IRKO in the Podo: impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy.Cell Metab 12, no. 4 (October 6, 2010): 311–12. https://doi.org/10.1016/j.cmet.2010.09.007.
Gurley, Susan B., and Thomas M. Coffman. “An IRKO in the Podo: impaired insulin signaling in podocytes and the pathogenesis of diabetic nephropathy.Cell Metab, vol. 12, no. 4, Oct. 2010, pp. 311–12. Pubmed, doi:10.1016/j.cmet.2010.09.007.
Journal cover image

Published In

Cell Metab

DOI

EISSN

1932-7420

Publication Date

October 6, 2010

Volume

12

Issue

4

Start / End Page

311 / 312

Location

United States

Related Subject Headings

  • Endocrinology & Metabolism
  • 3205 Medical biochemistry and metabolomics
  • 3101 Biochemistry and cell biology
  • 1101 Medical Biochemistry and Metabolomics
  • 0601 Biochemistry and Cell Biology