Cardiology
Publication
, Journal Article
Berger, JS; Becker, RC
May 5, 2010
This chapter summarizes the pathobiological mechanisms of coronary atherothrombosis, including, as a platform for understanding pharmacotherapies and evidence-based treatment strategies, its development, natural history and the clinical expression of disease. The classic model establishes platelets, and their complex interactions with inflammatory cells, activated endothelial cells, smooth muscle cells, apoptotic cells, oxidized LDL-cholesterol and coagulation proteins, at the epicenter of initiating events. © 2009 Blackwell Publishing Ltd.
Duke Scholars
Citation
APA
Chicago
ICMJE
MLA
NLM
Berger, J. S., & Becker, R. C. (2010). Cardiology, 185–193. https://doi.org/10.1002/9781444306286.ch18
Berger, J. S., and R. C. Becker. “Cardiology,” May 5, 2010, 185–93. https://doi.org/10.1002/9781444306286.ch18.
Berger JS, Becker RC. Cardiology. 2010 May 5;185–93.
Berger, J. S., and R. C. Becker. Cardiology. May 2010, pp. 185–93. Scopus, doi:10.1002/9781444306286.ch18.
Berger JS, Becker RC. Cardiology. 2010 May 5;185–193.