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Iron deficiency: lessons from anemic mice.

Publication ,  Journal Article
Andrews, NC
Published in: Yale J Biol Med
1997

Iron is an essential nutrient, and disorders of iron metabolism are common. Nonetheless, intestinal iron absorption and cellular iron transport are poorly understood. Biochemical approaches to elucidating these processes have yielded little in the past decade. As an alternative approach, we have begun to study spontaneous mouse mutants, that have inherited defects in key steps in iron transport. We have undertaken positional cloning of the gene responsible for microcytic anemia (gene symbol mk). This report describes the important characteristics of these mice, and our progress in studying them.

Duke Scholars

Published In

Yale J Biol Med

ISSN

0044-0086

Publication Date

1997

Volume

70

Issue

3

Start / End Page

219 / 226

Location

United States

Related Subject Headings

  • Mice, Mutant Strains
  • Mice
  • Male
  • Iron
  • Infant
  • Humans
  • Gastroenterology & Hepatology
  • Erythrocytes, Abnormal
  • Drug Resistance
  • Chromosome Mapping
 

Citation

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ICMJE
MLA
NLM
Andrews, N. C. (1997). Iron deficiency: lessons from anemic mice. Yale J Biol Med, 70(3), 219–226.
Andrews, N. C. “Iron deficiency: lessons from anemic mice.Yale J Biol Med 70, no. 3 (1997): 219–26.
Andrews NC. Iron deficiency: lessons from anemic mice. Yale J Biol Med. 1997;70(3):219–26.
Andrews, N. C. “Iron deficiency: lessons from anemic mice.Yale J Biol Med, vol. 70, no. 3, 1997, pp. 219–26.
Andrews NC. Iron deficiency: lessons from anemic mice. Yale J Biol Med. 1997;70(3):219–226.

Published In

Yale J Biol Med

ISSN

0044-0086

Publication Date

1997

Volume

70

Issue

3

Start / End Page

219 / 226

Location

United States

Related Subject Headings

  • Mice, Mutant Strains
  • Mice
  • Male
  • Iron
  • Infant
  • Humans
  • Gastroenterology & Hepatology
  • Erythrocytes, Abnormal
  • Drug Resistance
  • Chromosome Mapping