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MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer

Publication ,  Conference
Lamichhane, P; Nagathihalli, NS; Castellanos, J; Shi, C; Roberts, C; Vansaun, M; Merchant, N
Published in: PANCREAS
November 1, 2016

Duke Scholars

Published In

PANCREAS

EISSN

1536-4828

ISSN

0885-3177

Publication Date

November 1, 2016

Volume

45

Issue

10

Start / End Page

1518 / 1518

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Related Subject Headings

  • Gastroenterology & Hepatology
  • 3202 Clinical sciences
  • 1103 Clinical Sciences
 

Citation

APA
Chicago
ICMJE
MLA
NLM
Lamichhane, P., Nagathihalli, N. S., Castellanos, J., Shi, C., Roberts, C., Vansaun, M., & Merchant, N. (2016). MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer. In PANCREAS (Vol. 45, pp. 1518–1518). LIPPINCOTT WILLIAMS & WILKINS.
Lamichhane, P., N. S. Nagathihalli, J. Castellanos, C. Shi, C. Roberts, M. Vansaun, and N. Merchant. “MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer.” In PANCREAS, 45:1518–1518. LIPPINCOTT WILLIAMS & WILKINS, 2016.
Lamichhane P, Nagathihalli NS, Castellanos J, Shi C, Roberts C, Vansaun M, et al. MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer. In: PANCREAS. LIPPINCOTT WILLIAMS & WILKINS; 2016. p. 1518–1518.
Lamichhane, P., et al. “MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer.” PANCREAS, vol. 45, no. 10, LIPPINCOTT WILLIAMS & WILKINS, 2016, pp. 1518–1518.
Lamichhane P, Nagathihalli NS, Castellanos J, Shi C, Roberts C, Vansaun M, Merchant N. MEK Resistance via Amphiregulin Mediated EGFR-STAT3 Activation in Pancreatic Cancer. PANCREAS. LIPPINCOTT WILLIAMS & WILKINS; 2016. p. 1518–1518.

Published In

PANCREAS

EISSN

1536-4828

ISSN

0885-3177

Publication Date

November 1, 2016

Volume

45

Issue

10

Start / End Page

1518 / 1518

Publisher

LIPPINCOTT WILLIAMS & WILKINS

Related Subject Headings

  • Gastroenterology & Hepatology
  • 3202 Clinical sciences
  • 1103 Clinical Sciences